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Growth factors (Chur, Switzerland), 2005
Granulocyte macrophage-colony stimulating factor (GM-CSF) was originally defined by its ability to generate in vitro granulocyte and macrophage colonies from bone marrow precursor cells. Apart from its physiological role in the control of alveolar macrophage development, it now appears more likely that its major role lies in its ability to govern the ...
John A, Hamilton, Gary P, Anderson
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Granulocyte macrophage-colony stimulating factor (GM-CSF) was originally defined by its ability to generate in vitro granulocyte and macrophage colonies from bone marrow precursor cells. Apart from its physiological role in the control of alveolar macrophage development, it now appears more likely that its major role lies in its ability to govern the ...
John A, Hamilton, Gary P, Anderson
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International Journal of Antimicrobial Agents, 1998
M, Middleton, N, Thatcher
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M, Middleton, N, Thatcher
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Mechanism of GM-CSF stimulation of neutrophils
Immunologic Research, 1989Hemopoiesis, the development of functional blood cells, requires the action of colony-stimulating factors to promote the survival and stimulate the proliferation and differentiation of progenitor cells in the bone marrow [1-3]. Human granulocyte-macrophage colony-stimulating factor (GM-CSF) is a 22,000-dalton glycoprotein produced by T lymphocytes ...
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Emerging clinical uses for GM-CSF
European Journal of Cancer and Clinical Oncology, 1991variety early in life. Quitting smoking is associated with a dramatic drop in the risks of bladder cancer [7], irrespective of the type of tobacco. In the two studies which considered the effect of quitting separately by type, the relative risk remained well above the level of non-smokers only for smokers of black tobacco.
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Immunological Reviews, 2002
Summary: The crafting of genetic and biochemical techniques to identify cancer antigens yielded the unexpected discovery that immune recognition of tumors regularly accompanies cancer development. The failure of the host to suppress tumor formation or attenuate disease progression may thus reflect the limited immunogenicity of nascent tumors.
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Summary: The crafting of genetic and biochemical techniques to identify cancer antigens yielded the unexpected discovery that immune recognition of tumors regularly accompanies cancer development. The failure of the host to suppress tumor formation or attenuate disease progression may thus reflect the limited immunogenicity of nascent tumors.
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The choroid plexus links innate immunity to CSF dysregulation in hydrocephalus
Cell, 2023Emre Kızıltuğ +2 more
exaly