Results 311 to 320 of about 9,625,606 (401)
Graves' Disease and Microcytic Anemia: A Forgotten Connection. [PDF]
Daldal F, Dancis A.
europepmc +1 more source
The Mechanism of Postoperative Tetany in Graves' Disease
Tsukasa Murakami+4 more
openalex +2 more sources
Atrial Fibroblasts‐Derived Extracellular Vesicles Exacerbate Atrial Arrhythmogenesis
Exosome miR‐224‐5p derived from angiotensin II‐treated atrial fibroblasts creates a substrate for AF by promoting atrial electrical remodeling. Increased exosome miR‐224‐5p enhances AF susceptibility by inhibiting CACNA1c expression and decreasing ICa current of atrial cardiomyocytes.
Yue Yuan+13 more
wiley +1 more source
Serum metabolomics reveals systemic metabolic alterations of Graves' disease before and after antithyroid drug treatment. [PDF]
Li T+6 more
europepmc +1 more source
Genome-wide association study of male sexual orientation [PDF]
Cloninger, C. Robert+2 more
core +1 more source
This study presents a binary tumor‐microenvironment‐on‐a‐chip (T‐MOC) system incorporating multicellular tumor spheroids (MCTs) as an alternative preclinical platform to evaluate the efficacy of anticancer natural products. The T‐MOC model reproduces in vivo drug delivery barriers and physiological conditions, enabling morphological analysis to predict
Youngwon Kim+7 more
wiley +1 more source
Acute Pericarditis Associated With Graves' Disease: A Case Report. [PDF]
Estrela Santos M+4 more
europepmc +1 more source
FOXM1 maintains mitochondrial bioenergetic function by inhibiting MKRN1‐mediated ubiquitination of LKB1 in cardiomyocytes. Loss of FOXM1 in cardiomyocytes results in upregulation of MKRN1, which enhances LKB1 ubiquitination and disrupts AMPK signaling and energy metabolism pathways. Conversely, FOXM1 overexpression preserves mitochondrial bioenergetics
Shuai Song+17 more
wiley +1 more source