Results 21 to 30 of about 1,281,761 (360)

Frequent mild head injury promotes trigeminal sensitivity concomitant with microglial proliferation, astrocytosis, and increased neuropeptide levels in the trigeminal pain system. [PDF]

open access: yes, 2017
BACKGROUND: Frequent mild head injuries or concussion along with the presence of headache may contribute to the persistence of concussion symptoms. METHODS: In this study, the acute effects of recovery between mild head injuries and the frequency of ...
Assari, Soroush   +4 more
core   +2 more sources

Minor head injury [PDF]

open access: yesCurrent Opinion in Neurology, 1997
The relationship between brief loss of consciousness, subsequent cognitive and emotional complaints, and impact on daily functioning continues to be hotly debated. The weight of current evidence suggests that uncomplicated minor head injury can cause acute cognitive impairment that is organic in aetiology, but late onset or persisting symptoms result ...
D C, Macarthur, R F, Price
  +7 more sources

Admission to hospital following head injury in England: Incidence and socio-economic associations [PDF]

open access: yes, 2005
BACKGROUND: Head injury in England is common. Evidence suggests that socio-economic factors may cause variation in incidence, and this variation may affect planning for services to meet the needs of those who have sustained a head injury. METHODS:
A Carolei   +29 more
core   +3 more sources

Incidence of Mandibular Fractures Associated with Head Injury in Ninavah Governorate (2006-2007) [PDF]

open access: yesAl-Rafidain Dental Journal, 2011
Aims: The aims of the study are to find the incidences, types, sides and sites of mandibular fracture accompanied with head injuries in Ninavah Governorate. Materials and Methods: Data of trauma cases were collected from general, local, private hospitals
Ziad H Delemi , Mohammed K Hasouni
doaj   +1 more source

Hyperthermia following traumatic brain injury: a critical evaluation

open access: yesNeurobiology of Disease, 2003
Hyperthermia, frequently seen in patients following traumatic brain injury (TBI), may be due to posttraumatic cerebral inflammation, direct hypothalamic damage, or secondary infection resulting in fever.
Hilaire J Thompson   +4 more
doaj   +1 more source

Camel-related head injury in a high-income developing country

open access: yesJournal of Emergencies, Trauma and Shock, 2022
Introduction: Injury caused by large animals varies according to the regional distribution of the animals and their relationship to humans. Camels are usually friendly to humans; however, occasionally they become very hostile, especially in rutting ...
Ashraf F Hefny   +3 more
doaj   +1 more source

Implicit cognition is impaired and dissociable in a head-injured group with executive deficits [PDF]

open access: yes, 2005
Implicit or non-conscious cognition is traditionally assumed to be robust to pathology but Gomez-Beldarrain et al (1999, 2002) recently showed deficits on a single implicit task after head injury.
Andrade, J.   +4 more
core   +1 more source

modCHIMERA: A novel murine closed-head model of moderate traumatic brain injury [PDF]

open access: yes, 2018
Traumatic brain injury is a major source of global disability and mortality. Preclinical TBI models are a crucial component of therapeutic investigation.
Bayly, P. V.   +7 more
core   +2 more sources

Effect of Functional Electrical Stimulation combined with Treadmill Training and Challenging Tasks in a Patient with Stroke: A Case Report [PDF]

open access: yesJournal of Clinical and Diagnostic Research
Stroke is one of the leading causes of death globally. Advancements in medical science and technology have contributed to a reduction in the mortality rate among individuals who have experienced a stroke.
Deepanshi Srivastava   +4 more
doaj   +1 more source

The properties of nerve cell precursors in hydra [PDF]

open access: yes, 1986
Two signals, the head activator and an injury stimulus, control differentiation of nerve cells from uncommitted stem cells in hydra [Th. Holstein, H. C. Schaller, and C. N. David, (1986) Dev. Biol. 115, 9–17].
David, Charles N., Holstein, Thomas W.
core   +1 more source

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