Results 81 to 90 of about 13,894,413 (409)

Carcinoid Heart Disease

open access: yesJournal of the American College of Cardiology, 2010
![Figure][1] [![Graphic][3] ][3][![Graphic][4] ][4][![Graphic][5] ][5] A 26-year-old man with a history of carcinoid and small-bowel resection presented with shortness of breath.
Susie N. Hong   +2 more
openaire   +3 more sources

Heart to Heart, March 2013 [PDF]

open access: yes, 2013
Heart to Heart is a publication on new heart disease and stroke information and other related topics by the Department of Public ...

core  

Interdisciplinary three-step strategy to treat aortic stenosis and coronary artery disease in a patient with end-stage chronic obstructive pulmonary disease [PDF]

open access: yes, 2012
Background: Valvular aortic stenosis is a common disease in the elderly, often in multimorbid patients. It is often associated with coronary artery disease and peripheral artery disease.
Beiras Fernández, Andrés   +6 more
core   +2 more sources

Determination of ADP/ATP translocase isoform ratios in malignancy and cellular senescence

open access: yesMolecular Oncology, EarlyView.
The individual functions of three isoforms exchanging ADP and ATP (ADP/ATP translocases; ANTs) on the mitochondrial membrane remain unclear. We developed a method for quantitatively differentiating highly similar human ANT1, ANT2, and ANT3 using parallel reaction monitoring. This method allowed us to assess changes in translocase levels during cellular
Zuzana Liblova   +18 more
wiley   +1 more source

Loss of primary cilia promotes EphA2‐mediated endothelial‐to‐mesenchymal transition in the ovarian tumor microenvironment

open access: yesMolecular Oncology, EarlyView.
Loss of primary cilia in endothelial cells promotes EndMT and vascular abnormalities in the ovarian tumor microenvironment through EphA2 activation. Using human samples, in vitro models, and endothelial‐specific Kif3a‐knockout mice, we show that primary cilia loss drives the acquisition of cancer‐associated fibroblast‐like phenotypes, thereby ...
Jin Gu Cho   +11 more
wiley   +1 more source

Basic Mechanisms of Diabetic Heart Disease.

open access: yesCirculation Research, 2020
Diabetes mellitus predisposes affected individuals to a significant spectrum of cardiovascular complications, one of the most debilitating in terms of prognosis is heart failure.
R. Ritchie, E. Abel
semanticscholar   +1 more source

Heart Disease in Pregnancy

open access: yesSurgical Clinics of North America, 1957
Pregnancy imposes a significant and predictable circulatory burden which reaches a peak late in the second trimester. Heart failure is the major cause of maternal death in cardiac subjects, and the onset of decompensation and time of death are related to the hemodynamic burden of pregnancy.
openaire   +6 more sources

Evaluation and modification of tumor cell isolation techniques from malignant effusions for rapid drug sensitivity testing

open access: yesMolecular Oncology, EarlyView.
Non‐small cell lung cancer targeted treatment is limited to a few known genetic alterations, with few alternatives in advanced treatment lines. To direct treatment decisions by drug sensitivity testing (DST), this study compared several methods for tumor cell isolation from malignant effusions, pointing to repeated CD45+ cell depletion for effective ...
Navit Mooshayef   +10 more
wiley   +1 more source

Screening for coronary heart disease [PDF]

open access: yes, 1990
Malta and the United Kingdom share the dubious distinction of being among the three countries with the highest mortality rate from Cardiovascular disease in Europe; other countries in the Mediterranean basin have much lower rates. This presentation deals
Borg, Anton
core  

Profoundly reduced neovascularization capacity of bone marrow mononuclear cells derived from patients with chronic ischemic heart disease [PDF]

open access: yes, 2004
Background— Cell therapy with bone marrow–derived stem/progenitor cells is a novel option for improving neovascularization and cardiac function in ischemic heart disease.
Aicher, A   +8 more
core   +1 more source

Home - About - Disclaimer - Privacy