Results 221 to 230 of about 83,102 (265)
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Adding a Ventricle instead of transplanting a Heart

Nature, 1970
IN heart transplant operations, as practised at present, an unpaired vital organ is excised and another of doubtful viability and doubtful compatibility is substituted. It is no surprise to find that these operations are looked on with some disfavour and have a high mortality rate and poor long term results.
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The Right Ventricle in Congenital Heart Disease

Cardiology Clinics, 1992
An increasing number of children with congenital heart disease are surviving into adulthood, creating new, unusual patients with different physiologic and anatomic problems for the adult cardiologist. This article discusses those lesions affecting primarily the right ventricle.
Z M, Hijazi, W E, Hellenbrand
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Fat in the right ventricle of the normal heart

Histopathology, 2005
Aims : Fibrofatty replacement of the right ventricle wall, often with associated inflammation, is the hallmark of arrhythmogenic right ventricular cardiomyopathy (ARVC), a rare but established cause of sudden cardiac death in young adults. Fatty infiltration of the right ventricle alone without fibrosis may also occur but its relation to sudden death ...
D K, Tansey, Z, Aly, M N, Sheppard
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The Right Ventricle in Congenital Heart Diseases

2014
Right ventricular function is an important determinant of prognosis and outcome in congenital heart diseases. Right ventricular (RV) adaptation to congenital heart diseases (CHD) has many faces as there is a wide variety in defects involving the right ventricle as well as in treatment strategies.
Bartelds, Beatrijs, Berger, Rolf M. F.
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[Fibrillation of the heart ventricles].

Anesteziologiia i reanimatologiia, 1997
During ventricular fibrillation, extra feedback may form in the myocardial structure due to specific features of such fibrillation, specifically, because of highly asynchronous contractions of some parts of the myocardium; these newly forming feedback is conducive to a persistent development of such arrhythmia.
A M, Chernysh, M S, Bogushevich
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Calcium fluxes in frog heart ventricles

Pfl�gers Archiv European Journal of Physiology, 1969
Calcium fluxes in frog heart ventricles are determined both during quiescence and during periods of activity. The two main results: (a) calcium influx is much increased by activity; (b) a net release of calcium from the cells occurs immediately after activity, in parallel with, and probably related to, the decline in tension of the downward “staircase”.
R, Niedergerke, S, Page, M S, Talbot
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Lipid composition of beef heart ventricle

Lipids, 1967
AbstractThe lipid class composition of beef heart ventricle was determined by a combination of diethylaminoethyl cellulose column chromatography and quantitative thin‐layer chromatography. Percentages of the total lipid were: triglyceride, 43.6; cholesterol, 7.4; phosphatidyl choline, 22.8; phingomyelin, 4.0; phosphatidyl ethanolamine, 11.2 ...
M L, Das, G, Rouser
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Sodium Exchange in the Frog Heart Ventricle

American Journal of Physiology-Legacy Content, 1957
The isolated frog heart ventricle was used to study the kinetics of uptake and release of radioactive sodium. Sucrose was used as an extracellular space indicator. A flux of sodium across the ventricle cell membranes of 15 x 10–12 m/cm2 sec. was calculated from the data.
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Actomyosin content of rabbit heart ventricle

American Journal of Physiology-Legacy Content, 1964
The requirements for quantitative extraction of rabbit cardiac actomyosin were investigated. Extraction of the tissue was carried out at pH 6.5. Under these conditions, precipitation of actomyosin from the soluble tissue extract showed a plateau between pH 6.9 and 7.1.
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[Diastolic filling of the heart ventricle].

Kardiologiia, 1983
It is demonstrated that negative, "subatmospheric" pressure developing in the heart ventricles during the rapid-filling phase is an evidence of active diastole and inflated capacity of heart chambers. Diastolic activity of the heart increases as a result of insufficient blood inflow to the ventricles coupled with inadequate venous return or narrowed ...
V I, Astaf'ev   +3 more
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