Results 201 to 210 of about 98,569 (252)
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Immune Hemolytic Anemia

Medical Clinics of North America, 1980
Immune hemolytic anemia is an acquired anemia resulting from the premature destruction of red cells caused by the presence of antibody and/or complement on the red cell surface. The Coombs test, modified and improved, remains the mainstay of diagnosis.
J A, Axelson, A F, LoBuglio
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Autoimmune Hemolytic Anemias

New England Journal of Medicine, 2021
Autoimmune Hemolytic Anemia Hemolytic anemias, which are rare, are often caused by autoimmune destruction of red cells. The hemolysis can be intravascular or extravascular.
Sigbjørn Berentsen, Wilma Barcellini
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Autoimmune Hemolytic Anemia

Medical Clinics of North America, 2017
Autoimmune hemolytic anemia is an acquired autoimmune disorder resulting in the production of antibodies directed against red blood cell antigens causing shortened erythrocyte survival. The disorders can present as a primary disorder (idiopathic) or secondary to other autoimmune disorders, malignancies, or infections.
Howard A, Liebman, Ilene C, Weitz
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Autoimmune hemolytic anemia

The Indian Journal of Pediatrics, 2008
To study the clinico-hematological profile and treatment outcome in children suffering from auto immune hemolytic anemia (AIHA).Twelve children were diagnosed with auto immune hemolytic anemia over a period of four years. Direct antiglobulin test was positive in all the cases.
V, Gupta, J, Shukla, B D, Bhatia
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Congenital Hemolytic Anemia

Medical Clinics of North America, 2017
Red blood cell (RBC) destruction can be secondary to intrinsic disorders of the RBC or to extrinsic causes. In the congenital hemolytic anemias, intrinsic RBC enzyme, RBC membrane, and hemoglobin disorders result in hemolysis. The typical clinical presentation is a patient with pallor, anemia, jaundice, and often splenomegaly.
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Autoimmune Hemolytic Anemia

Pediatric Clinics of North America, 1980
In autoimmune hemolytic anemia, individuals produce antibodies directed against one of their own erythrocyte membrane antigens. The hemolysis in autoimmune hemolytic anemia is most commonly extravascular rather than intravascular, and the liver and spleen play a major role in the clearance of the antibody-coated cells.
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Autoimmune Hemolytic Anemia

Archives of Internal Medicine, 1975
Warm-type autoantibodies of autoimmune hemolytic anemia (AIHA) are usually IgG but may be IgM or IgA. They are usual Rh specific. Cold-type antibodies are IgM or IgG (Donath-Landsteiner [DL] antibody). IgM antibodies are usually anit-l (occasionally anti-i) and DL antibodies anti-P.
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Tolbutamide-induced Hemolytic Anemia

Diabetes, 1977
A 67-year-old female diabetic is presented who developed a Coombs'-positive hemolytic anemia after a year of treatment with tolbutamide. An IgG antibody was identified in the patient's serum that caused the agglutination of both the patient's red blood cells and tolbutamide-coated erythrocytes in the absence of complement. Such a reaction did not occur
P, Malacarne   +3 more
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Microangiopathic Hemolytic Anemia

New England Journal of Medicine, 1969
IN 1917 Rous and Robertson1 suggested that fragmentation was the normal fate of the effete erythrocyte at the end of its life-span. More recently it has become recognized that erythrocytes may undergo fragmentation earlier if they are subjected to excessive trauma within the circulation.
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Autoimmune hemolytic anemia

Human Pathology, 1983
Nowhere in the management of patients with autoimmune hemolytic anemias is the communication between clinician and laboratory personnel more important than in regard to blood transfusion. A clinical decision that blood transfusion is necessary must be tempered by the knowledge that transfusion has a greater-than-usual risk in this setting, both because
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