Results 291 to 300 of about 10,599,387 (354)
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Pathogenesis of ABO hemolytic disease
American Journal of Obstetrics and Gynecology, 1960Abstract 1.1. Observations on 51 families in which ABO hemolytic disease occurred are reviewed. In all but one of the families, the mothers were found to be Group O in conformity with previous reports. This is attributed to the ability of Group O mothers to form antibodies of specificity anti-C as well as anti-A and anti-B. 2.2.
A S, WIENER +3 more
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Postgraduate Medicine, 1966
Acquired hemolytic disease (AHD) is an auto-immune disease characterized primarily by a shortened life-span and rapid regeneration of red cells. Idiopathic or secondary AHD is suspected any time a chronically anemic patient also has a chronically elevated reticulocyte count. It is diagnosed by demonstrating the immune mechanism by the antiglobulin test
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Acquired hemolytic disease (AHD) is an auto-immune disease characterized primarily by a shortened life-span and rapid regeneration of red cells. Idiopathic or secondary AHD is suspected any time a chronically anemic patient also has a chronically elevated reticulocyte count. It is diagnosed by demonstrating the immune mechanism by the antiglobulin test
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Hemochromatosis and Hemolytic Disease
Archives of Internal Medicine, 1980Many hypotheses, most of them wrong, deal with the duodenum and iron absorption. At present, we accept that the balance of iron between the organism and its environment is controlled not by excretion of inadvertent excess but by the intestine's intelligent restriction of the amount it absorbs from dietary sources.
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ABO Hemolytic Disease of the Newborn
American Journal of Clinical Pathology, 1979The charts of newborn infants with positive direct Coombs' test were studied. Only cases in which the mother's blood was group O and the infant's group A or group B were studied. There was no difference between group A and group B infants in the frequency and severity of the hemolytic process caused by maternal antibodies.
S Y, Chan-Shu, O, Blair
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Autoimmune Hemolytic Disease in the Dog
Journal of the American Veterinary Medical Association, 1971SUMMARY A 10-year-old Welsh Terrier bitch with idiopathic autoimmune hemolytic anemia has been observed and treated for 2 years. During periods of remission and relapse there were marked differences in morphologic features, osmotic fragility, and autologous survival of red blood cells (rbc), whereas the direct antiglobulin reaction remained uniformly ...
R W, Bull, R, Schirmer, A J, Bowdler
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Hemolytic Disease of the Newborn
Postgraduate Medicine, 1966Management of the erythroblastotic infant in iso-immunized pregnancies has progressed from simple exchange transfusion in 1945 through early delivery and amniotic fluid examination to the latest innovation, intraperitoneal fetal transfusion. With each improvement infant mortality has decreased, as is shown in the records of the Rh Laboratory, Winnipeg.
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Texas Journal of Medical Science, 2022
Hemolytic anemia is a condition where red blood cells disappear faster than they can be produced. The destruction of red blood cells is called hemolysis.
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Hemolytic anemia is a condition where red blood cells disappear faster than they can be produced. The destruction of red blood cells is called hemolysis.
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Serum Haptoglobin in Hemolytic Disease
JAMA: The Journal of the American Medical Association, 1980To the Editor.— Inadequate construction of study and control groups by Marchand et al (1980;243:1909) inflates the apparent sensitivity and specificity of their serum haptoglobin assay. More important, there is no reason to believe that the test has much practical value.
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Seminars in hematology (Print), 2018
The classical complement pathway and, to some extent, the terminal pathway, are involved in the immune pathogenesis of autoimmune hemolytic anemia (AIHA).
S. Berentsen
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The classical complement pathway and, to some extent, the terminal pathway, are involved in the immune pathogenesis of autoimmune hemolytic anemia (AIHA).
S. Berentsen
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Hemolytic Anemia in Wilson's Disease
Annals of Internal Medicine, 1970Abstract Penicillamine therapy was discontinued for a period of 200 days in a patient with Wilson's disease. Copper reaccumulated in the body at the rate of about 0.43 mg/day.
A, Deiss, G R, Lee, G E, Cartwright
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