Results 41 to 50 of about 17,173 (205)

DE PICASSO A PEDRO AMÉRICO: ENSINANDO A HEMOSTASIA SECUNDÁRIA ATRAVÉS DE PINTURAS CLÁSSICAS

open access: yesHematology, Transfusion and Cell Therapy
Introdução: O ensino da fisiologia da hemostasia é um desafio nos cursos de graduação de saúde. Por ser um tema de natureza eminentemente teórico com grande densidade de conteúdo e necessidade de memorização, há uma natural aversão pela maioria dos ...
LFB Botelho
doaj   +1 more source

Didactics for the teaching-learning processes in the training practice in hemostasia [PDF]

open access: yes, 2017
El desarrollo de pruebas diagnósticas para la detección de alteraciones de la hemostasia, su interpretación y correlación, requieren del estudio permanente y detallado de los procesos bioquímicos, fisiológicos y patológicos.
Reina Soler, Aura Milena
core  

Efeito da hemodiluição normovolêmica aguda na coagulação sanguínea: comparação entre os testes colhidos de um modelo in vivo e de um modelo in vitro Efecto de la hemodilución normovolémica aguda en la coagulación sanguínea: comparación entre los tests recolectados en un modelo in vivo y en un modelo in vitro Effects of acute normovolemic hemodilution on blood coagulation: comparison between tests of an in vivo and an in vitro model

open access: yesRevista Brasileira de Anestesiologia, 2010
JUSTIFICATIVA E OBJETIVOS: A hemodiluição normovolêmica produz resultados conflitantes na hemostasia, pois os trabalhos diferem quanto a tipo de líquido utilizado, profundidade da hemodiluição, método utilizado para avaliar a hemostasia e forma de se ...
Marco Aurélio Beloto de Souza   +2 more
doaj   +1 more source

A review of the blood coagulation system of fish. [PDF]

open access: yes, 2018
Uma revisão do sistema de coagulação sanguínea de peixes. Hemostasia é uma função de grande importância para responder a danos e desordens da coagulação sanguínea.
OLIVEIRA, S. R., TAVARES-DIAS, M.
core  

Role of Inflammatory and Proresolving Mediators in Endothelial Dysfunction

open access: yesBasic &Clinical Pharmacology &Toxicology, Volume 136, Issue 5, May 2025.
ABSTRACT Excessive local inflammation is a common mechanism in many cardiovascular diseases (CVDs) such as hypertension, atherosclerosis and aortic aneurysms. In endothelial cells, inflammatory cytokines such as interferons, tumour necrosis factor alpha or interleukins increase oxidative stress and contractile prostanoids and the expression of adhesion
Ana M. Briones   +7 more
wiley   +1 more source

Manejo perioperatorio de fármacos anticoagulantes y antiagregantes en el paciente con fractura de cadera [PDF]

open access: yes, 2016
La fractura de cadera eleva de manera significativa en los estudios publicados la morbilidad y la mortalidad en los pacientes ancianos que la padecen. La participación en el proceso de fármacos que alteran la hemostasia se ha convertido en uno de los ...
Aguar Carrascosa, Pablo   +6 more
core  

Pharmacokinetics of statins [PDF]

open access: yes, 2005
Statins are lipid lowering agents that promote their effects on plasma lipids through the inhibition of HMG-CoA reductase, a crucial enzyme in the cascade of cholesterol synthesis, leading to reduction of tissue cholesterol pool and consequently, to an ...
Fonseca, Francisco Antonio Helfenstein
core   +4 more sources

How to explain the beneficial effects of leukocyte‐ and platelet‐rich fibrin

open access: yesPeriodontology 2000, Volume 97, Issue 1, Page 74-94, February 2025.
Abstract The survival of an organism relies on its ability to repair the damage caused by trauma, toxic agents, and inflammation. This process involving cell proliferation and differentiation is driven by several growth factors and is critically dependent on the organization of the extracellular matrix. Since autologous platelet concentrates (APCs) are
Juan Blanco   +3 more
wiley   +1 more source

Cystathionine β-synthase and methylenetetrahydrofolate reductase mutations in Mexican individuals with hyperhomocysteinemia

open access: yesSAGE Open Medicine, 2020
Background: Hyperhomocysteinemia, a thrombotic risk factor, may have several causes. Among the genetic causes of hyperhomocysteinemia, there are polymorphisms in the enzymes methylenetetrahydrofolate reductase (C677T) and cystathionine β-synthase (C699T,
Anahi Guadalupe Figueroa-Torres   +10 more
doaj   +1 more source

A phase 1/2 safety and efficacy study of TAK‐754 gene therapy: The challenge of achieving durable factor VIII expression in haemophilia A clinical trials

open access: yesHaemophilia, Volume 31, Issue 1, Page 108-117, January 2025.
Abstract Introduction Haemophilia A is an X‐linked bleeding disorder resulting from a deficiency of factor VIII (FVIII). To date, multiple gene therapies have entered clinical trials with the goal of providing durable haemostatic protection from a single dose. TAK 754 (BAX 888) is an investigational AAV8‐based gene therapy containing a FVIII transgene.
John Chapin   +13 more
wiley   +1 more source

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