Results 101 to 110 of about 209,459 (341)
Relationship of Platelet Serotonin to Disturbances of Clotting and Hemostasis [PDF]
Murray Weiner, Sidney Udenfriend
openalex +1 more source
An ideal implant should mimic native tissues such that it can integrate, sense, heal, and continue to function, i.e., be autonomous. Although early, there are good steps taken in this way, e.g., the development of stimuli‐responsive, self‐powering, self‐actuating, self‐healing, self‐regenerating, and self‐aware implants.
Jagan Mohan Dodda +5 more
wiley +1 more source
Current guidelines on treatment of metastatic renal cell carcinoma (mRCC) suggest an emerging role of local therapy (LT). Still there is a lack of data which patients may benefit from additional LT once medical treatment (MT) is initiated.
Sena Hoffer +10 more
doaj +1 more source
CD40L Priming of Platelets via NF‐κB Activation is CD40‐ and TAK1‐Dependent
Background CD40 ligand (CD40L) is a thromboinflammatory molecule that predicts cardiovascular events. CD40L is a strong activator of nuclear factor kappa B (NF‐κB) in platelets that primes and enhances platelet activation in response to thrombotic ...
Kevin Kojok +4 more
doaj +1 more source
Ultrasound‐Powered, Battery‐Free Implants via Triboelectric Energy Harvesting
Ultrasound‐driven triboelectric nanogenerators (US‐TENGs) present a promising approach to battery‐free implants by combining body‐safe ultrasound with the material and structural versatility of TENGs. This review categorizes current US‐TENG research into long‐term and transient short‐term applications and discusses key challenges in materials, device ...
Youngwook Chung +5 more
wiley +1 more source
Background: The pTα/preTCR regulates the β-selection, a crucial T-cell developmental checkpoint, providing a most potent survival advantage to thymocytes mediated by the src-kinase p56Lck.
Philipp Ivanyi +6 more
doaj +1 more source
Hemostasis and Hemotherapy [PDF]
W. Korte, M. Cattaneo
openaire +3 more sources
This study uncovers a novel mechanism and therapeutic targets in obstetric antiphospholipid syndrome (OAPS). Antiphospholipid antibody/ß2‐glycoprotein I complex boosts CCL2 and TNF‐α expression in decidual macrophages via TLR4‐NF‐κB. CCL2 accumulates on endothelial surfaces via ACKR1, recruiting monocyte‐derived CCR2+ macrophages, driving inflammation,
Rui Gao +13 more
wiley +1 more source

