Results 61 to 70 of about 11,309 (209)

Flying-fox species density--a spatial risk factor for Hendra virus infection in horses in eastern Australia. [PDF]

open access: yesPLoS ONE, 2014
Hendra virus causes sporadic but typically fatal infection in horses and humans in eastern Australia. Fruit-bats of the genus Pteropus (commonly known as flying-foxes) are the natural host of the virus, and the putative source of infection in horses ...
Craig Smith   +4 more
doaj   +1 more source

Climate Anomalies and Spillover of Bat-Borne Viral Diseases in the Asia–Pacific Region and the Arabian Peninsula

open access: yesViruses, 2022
Climate variability and anomalies are known drivers of the emergence and outbreaks of infectious diseases. In this study, we investigated the potential association between climate factors and anomalies, including El Niño Southern Oscillation (ENSO) and ...
Alice Latinne, Serge Morand
doaj   +1 more source

Real‐time fluorometric isothermal assays for detection of Streptococcus equi subspecies equi and Streptococcus equi subspecies zooepidemicus in horses: Validation, comparison and evaluation of their clinical application

open access: yesAustralian Veterinary Journal, Volume 104, Issue 3, Page 158-166, March 2026.
Infectious diseases significantly impact equine health and welfare, causing illness and death, and loss of productivity globally. One such disease is ‘strangles’, a highly contagious upper respiratory condition in horses caused by Streptococcus equi subspecies equi (SEE).
M Jelocnik   +11 more
wiley   +1 more source

Evidence of endemic Hendra virus infection in flying-foxes (Pteropus conspicillatus)--implications for disease risk management. [PDF]

open access: yesPLoS ONE, 2011
This study investigated the seroepidemiology of Hendra virus in a spectacled flying-fox (Pteropus conspicillatus) population in northern Australia, near the location of an equine and associated human Hendra virus infection in late 2004.
Andrew C Breed   +3 more
doaj   +1 more source

Plant phenology supports the multi-emergence hypothesis for ebola spillover events [PDF]

open access: yes, 2017
Ebola virus disease outbreaks in animals (including humans and great apes) start with sporadic host switches from unknown reservoir species. The factors leading to such spillover events are little explored.
Douglas, Noah E.   +6 more
core   +1 more source

Skin Colour in Salamanders Is Modulated by Both Epitranscriptomic Methylation and Gene Expression

open access: yesMolecular Ecology, Volume 35, Issue 6, March 2026.
ABSTRACT Animal colouration is a key trait in organismal biology, being involved in natural and sexual selection, competition, and communication. Amphibians use their highly diverse colouration in many ecological interactions, but the molecular bases of their colour variation are less well understood than in other vertebrate systems. While the genetic,
Nicholas Strowbridge   +3 more
wiley   +1 more source

ZNRD2 Mediated Nucleoprotein Aggregation Impairs Respiratory Syncytial Virus Replication

open access: yesAdvanced Science, Volume 13, Issue 12, 27 February 2026.
During RSV infection, nucleoprotein (N) forms RNA‐bound oligomers. The host protein ZNRD2 binds to these oligomers, promoting their transition into insoluble aggregates. These aggregates simultaneously sequester functional N to restrict viral production and disrupt chaperonin assembly quality control by interfering with ZNRD2's role as an adaptor ...
Haiwu Zhou   +8 more
wiley   +1 more source

No Evidence of Prolonged Hendra Virus Shedding by 2 Patients, Australia

open access: yesEmerging Infectious Diseases, 2012
To better understand the natural history of Hendra virus infection and its tendency to relapse, 2 humans infected with this virus were monitored after acute infection.
Carmel Taylor   +4 more
doaj   +1 more source

EPI Update, October 7 2005 [PDF]

open access: yes, 2005
Weekly newsletter for Center For Acute Disease Epidemiology of Iowa Department of Public ...

core  

Ephrin-B2 expression critically influences Nipah virus infection independent of its cytoplasmic tail [PDF]

open access: yes, 2008
Background Cell entry and cell-to-cell spread of the highly pathogenic Nipah virus (NiV) requires binding of the NiV G protein to cellular ephrin receptors and subsequent NiV F-mediated fusion.
Augustin Hellmut G   +5 more
core   +2 more sources

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