Results 1 to 10 of about 8,714 (218)

Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report [PDF]

open access: yesJournal of Medical Case Reports, 2011
Introduction Henoch-Schönlein purpura is the most common systemic vasculitis in children. Typical presentations are palpable purpura, abdominal pain, arthritis, and hematuria.
Howarth Charles B   +3 more
doaj   +2 more sources

Gastrointestinal Biopsy in Henoch-Schönlein Purpura: A Great Diagnostic Contribution [PDF]

open access: yesEuropean Journal of Case Reports in Internal Medicine, 2017
Henoch-Schönlein purpura is an IgA-mediated immune vasculitis which is characterized by purpuric lesions and osteoarticular, intestinal and sometimes renal manifestations. The histopathological substrate of this entity is leucocytoclastic vasculitis (LCV)
Imen Akkari   +2 more
doaj   +4 more sources

Henoch–Schönlein purpura induced by sitagliptin: A case report [PDF]

open access: yesSAGE Open Medical Case Reports
Dipeptidyl peptidase-4 inhibitors are oral antihyperglycemic medications often used as adjuncts to other antidiabetic agents to treat type 2 diabetes mellitus.
Alexa Moschella, Carly Kirshen
doaj   +2 more sources

Hemorrhagic Intestinal Henoch-Schonlein Purpura Complicated by Cytomegalovirus Infection

open access: yesCanadian Journal of Gastroenterology & Hepatology, 1998
A 54-year-old man on hemodialysis for acute chronic renal failure and on corticosteroids for Henoch-Schonlein purpura developed massive hematochezia. After extensive clinical investigation, an ileal bleeding site was identified and surgically removed ...
Laurence D Jewell, A B R Thomson
exaly   +2 more sources

Lumbar swelling and migrating edema in 3- and 4-year-old boys

open access: yesSAGE Open Medical Case Reports, 2022
Henoch-Schönlein purpura is the most common systemic vasculitis in children, characterized by IgA deposits in small vessels. The etiology is unknown, but Henoch-Schönlein purpura typically follows an upper respiratory infection, or less frequently other ...
Marta Marcia, Emilia Parodi
doaj   +1 more source

Acute Epstein-Barr Virus Hepatitis in a 32 Month Old Female Manifesting as Henoch-Schönlein Purpura [PDF]

open access: yesChildhood Kidney Diseases, 2015
Henoch-Schönlein purpura can result from exposure to an antigen after infection with several types of organisms. However, Henoch-Schönlein purpura caused by a primary Epstein-Barr virus infection has been rarely reported. Here, we report the case of a 32-
Hee Jin Kim, Su Jin Jung, Jun Ho Lee
doaj   +1 more source

Anesthetic Management of A Patient with Henoch-Schonlein Purpura for Caesarean Section

open access: yesIndonesian Journal of Anesthesiology and Reanimation (IJAR), 2022
Introduction: Henoch-Schonlein Purpura or Immunoglobulin-A vasculitis is a systemic vasculitis caused by immune complexes that attack small blood vessels.
Indriyani Wijaya
doaj   +1 more source

Risk factors for renal involvement in Henoch–Schönlein purpura

open access: yesJornal de Pediatria, 2021
Objective: Henoch–Schönlein purpura is a systemic vasculitis that mainly occurs in children. Renal impairment is a major complication of Henoch–Schönlein purpura, but there is no established predictive marker for renal involvement.
Woo Kyung Kim   +2 more
doaj   +1 more source

Henoch–Schönlein Purpura (IgA Vasculitis) in Association with Thyrotoxicosis

open access: yesCase Reports in Endocrinology, 2021
Graves’ disease is the most common cause of hyperthyroidism, which is characterized by thyroid antibodies and the following clinical manifestations: goiter, ophthalmopathy, and pretibial myxedema.
Mojgan Sanjari   +2 more
doaj   +1 more source

Henoch-Schönlein purpura with acute pancreatitis: analysis of 13 cases

open access: yesBMC Pediatrics, 2018
Background Henoch-Schönlein purpura is a common small vessel vasculitis in children. Acute pancreatitis rarely presents as a complication of Henoch-Schönlein purpura and has not been well characterized.
Qin Zhang   +6 more
doaj   +1 more source

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