Results 311 to 320 of about 269,557 (334)
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Frontiers in Bioscience, 2002
Herpes simplex virus (HSV) commonly causes human infections in the orofacial region (HSV-1) and in the genital region (HSV-2). Productive viral infection in mucosal epithelial cells may result in clinical symptoms and is followed by a latent infection within sensory neurons.
Travis J, Taylor +3 more
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Herpes simplex virus (HSV) commonly causes human infections in the orofacial region (HSV-1) and in the genital region (HSV-2). Productive viral infection in mucosal epithelial cells may result in clinical symptoms and is followed by a latent infection within sensory neurons.
Travis J, Taylor +3 more
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Infection Control, 1986
Herpes simplex virus (HSV) causes a variety of illnesses in th e community and hospital settings (Table). Nosocomial infections with this virus may result from: 1) reactivation of latent infection, especially in patients whose immune systems are compromised; 2) spread from mother to infant; 3) spread from patients to hospital personnel; 4) spread from ...
B P, Simmons, M S, Gelfand
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Herpes simplex virus (HSV) causes a variety of illnesses in th e community and hospital settings (Table). Nosocomial infections with this virus may result from: 1) reactivation of latent infection, especially in patients whose immune systems are compromised; 2) spread from mother to infant; 3) spread from patients to hospital personnel; 4) spread from ...
B P, Simmons, M S, Gelfand
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Survey of Ophthalmology, 1977
This article reviews the current concepts for the diagnosis and treatment of Herpes simplex keratitis and its complications, discussing characteristics of the virus, the prevalence of the disease, chemical and clinical differentiation between Types I and II Herpesvirus hominis, and the many clinical presentations of Type I disease. The antiviral agents
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This article reviews the current concepts for the diagnosis and treatment of Herpes simplex keratitis and its complications, discussing characteristics of the virus, the prevalence of the disease, chemical and clinical differentiation between Types I and II Herpesvirus hominis, and the many clinical presentations of Type I disease. The antiviral agents
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Pediatrics in Review, 2004
1. Linda A. Waggoner-Fountain, MD* 2. Leigh B. Grossman, MD† 1. *Assistant Professor of Pediatrics, Division of Infectious Diseases 2. †Professor of Pediatrics and Head, Division of Infectious Diseases, University of Virginia School of Medicine, Charlottesville, VA After completing this article, readers should be able to: 1.
Linda A, Waggoner-Fountain +1 more
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1. Linda A. Waggoner-Fountain, MD* 2. Leigh B. Grossman, MD† 1. *Assistant Professor of Pediatrics, Division of Infectious Diseases 2. †Professor of Pediatrics and Head, Division of Infectious Diseases, University of Virginia School of Medicine, Charlottesville, VA After completing this article, readers should be able to: 1.
Linda A, Waggoner-Fountain +1 more
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Australian and New Zealand Journal of Medicine, 1977
In this paper seven cases of severe viral encephalitis seen over a four year period at the Princes Alexandria Hospital, Brisbane, are presented. Four of these cases were proven cases of Herpes simplex encephalitis on the basis of viral culture, and three were probable cases of Herpes simplex encephalitis on the basis of rising serum antibody titres.
R S, Boyle, P J, Landy
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In this paper seven cases of severe viral encephalitis seen over a four year period at the Princes Alexandria Hospital, Brisbane, are presented. Four of these cases were proven cases of Herpes simplex encephalitis on the basis of viral culture, and three were probable cases of Herpes simplex encephalitis on the basis of rising serum antibody titres.
R S, Boyle, P J, Landy
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Ophthalmology, 1983
Avirulent strains of herpes simplex type 1 (HSV-1) infect and shed and colonize the ganglia in rabbits. This primary infection reduces the severity of subsequent infection by virulent virus and prevents ganglionic colonization by even very virulent neurotropic virus.
H E, Kaufman +2 more
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Avirulent strains of herpes simplex type 1 (HSV-1) infect and shed and colonize the ganglia in rabbits. This primary infection reduces the severity of subsequent infection by virulent virus and prevents ganglionic colonization by even very virulent neurotropic virus.
H E, Kaufman +2 more
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Progress in Retinal and Eye Research, 2006
Herpes simplex keratitis (HSK) results from an infection with the herpes simplex virus type 1 (HSV-1) also known as human herpesvirus type 1 (HHV-1). Primary infection may involve an ocular or non-ocular site, following which latency might be established principally in the trigeminal ganglion but also in the cornea. During latency, the virus appears as
Stephen, Kaye, Anshoo, Choudhary
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Herpes simplex keratitis (HSK) results from an infection with the herpes simplex virus type 1 (HSV-1) also known as human herpesvirus type 1 (HHV-1). Primary infection may involve an ocular or non-ocular site, following which latency might be established principally in the trigeminal ganglion but also in the cornea. During latency, the virus appears as
Stephen, Kaye, Anshoo, Choudhary
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Herpes Simplex Keratoconjunctivitis
American Journal of Ophthalmology, 1966A 29-year-old mechanical engineer was seen in the Yale-New Haven Emergency Room with painful eyes, malaise, chills and fever. Five days prior to admission, a small quantity of turpentine had splashed into his left eye while he was wear ing a contact lens.
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Archives of Ophthalmology, 1978
Fatal encephalitis with accompanying retinitis developed in a previously healthy 18-month-old infant. Clinically the disease appeared as whitish-yellow punctate lesions, perivascular cuffing, and hemorrhage. The antibody titer to herpes simplex rose from 1:8 on the day of admission to 1:256 on the day of death. Postmortem, intranuclear inclusion bodies
G W, Cibis, J T, Flynn, E B, Davis
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Fatal encephalitis with accompanying retinitis developed in a previously healthy 18-month-old infant. Clinically the disease appeared as whitish-yellow punctate lesions, perivascular cuffing, and hemorrhage. The antibody titer to herpes simplex rose from 1:8 on the day of admission to 1:256 on the day of death. Postmortem, intranuclear inclusion bodies
G W, Cibis, J T, Flynn, E B, Davis
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1993
Herpes simplex infections may cause significant morbidity following bone marrow transplantation (BMT) (Saral 1988). The pathophysiology of these infections was studied in the pre-antiviral era and provided the basis for the development of studies designed to evaluate the efficacy of promising antiviral agents.
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Herpes simplex infections may cause significant morbidity following bone marrow transplantation (BMT) (Saral 1988). The pathophysiology of these infections was studied in the pre-antiviral era and provided the basis for the development of studies designed to evaluate the efficacy of promising antiviral agents.
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