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HHV-6-positivity in diseases with demyelination
Journal of Clinical Virology, 2014The triggering agent of multiple sclerosis is still unknown and many viruses, including human herpesvirus-6 (HHV-6), are under suspicion. In earlier study we found patients who had HHV-6 reactive OCBs in their CSF. We wanted to investigate whether HHV-6 has an active role in diseases with demyelination.To analyze the HHV-6-reactive cases in detail and ...
Oili Salonen +5 more
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2006
Publisher Summary This chapter discusses the body of knowledge accrued over the last two decades on the biology of human herpesvirus-6 (HHV-6). HHV-6 has a restricted range of susceptible species, essentially limited to humans and selected nonhuman primates.
Lorenzo Dagna +2 more
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Publisher Summary This chapter discusses the body of knowledge accrued over the last two decades on the biology of human herpesvirus-6 (HHV-6). HHV-6 has a restricted range of susceptible species, essentially limited to humans and selected nonhuman primates.
Lorenzo Dagna +2 more
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Case of fatal encephalitis by HHV‐6 variant A
Journal of Medical Virology, 2001AbstractA fatal case is reported of encephalitis in an 85‐year‐old man caused by the human herpesvirus 6 variant A. The virological diagnosis was based on the findings of the virus variant genomic sequences both in the cerebrospinal fluid and serum of the patient. Moreover, virus replication in nervous tissue was suggested by a viral load higher in the
PORTOLANI, Marinella +5 more
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Nature Medicine, 1997
Viruses have long been suggested to be involved in the etiology of multiple sclerosis (MS). This suggestion is based on (1) epidemiological evidence of childhood exposure to infectious agents and increase in disease exacerbations with viral infection; (2) geographic association of disease susceptibility with evidence of MS clustering; (3) evidence that
SOLDAN SS +11 more
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Viruses have long been suggested to be involved in the etiology of multiple sclerosis (MS). This suggestion is based on (1) epidemiological evidence of childhood exposure to infectious agents and increase in disease exacerbations with viral infection; (2) geographic association of disease susceptibility with evidence of MS clustering; (3) evidence that
SOLDAN SS +11 more
openaire +2 more sources
2006
Publisher Summary A prospective population-based study was recently undertaken to determine the clinical features of primary human herpesvirus-6 (HHV-6) infection outside the acute-care setting. Several clinical conditions such as skin rash resembling acute graft-versus-host-disease (GVHD), bone marrow suppression, interstitial pneumonitis, and ...
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Publisher Summary A prospective population-based study was recently undertaken to determine the clinical features of primary human herpesvirus-6 (HHV-6) infection outside the acute-care setting. Several clinical conditions such as skin rash resembling acute graft-versus-host-disease (GVHD), bone marrow suppression, interstitial pneumonitis, and ...
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HHV-6 and the Cardiovascular System
2006Publisher Summary This chapter reviews a case of an adult patient who developed fulminant myocarditis after steroid pulse therapy for acute hepatitis. This case provides further insights into the mechanisms of virus–myocardium–immune interactions. A person was referred to a hospital because of an exacerbation of fulminant myocarditis after undergoing
Satoki Fukae, Naoto Ashizawa
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HHV-6 and the Respiratory System
2006Publisher Summary Various inflammatory reactions occur in the respiratory tract, both in primary and in non-primary infections with human herpesvirus-6 (HHV-6). Pneumonia following primary HHV-6 infection is rare, yet when occurring, it will usually require hospital admission of the seriously ill child. It occurs during the initial phase of infection,
Wiersbitzky H +2 more
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2006
Publisher Summary Human herpesvirus-6 (HHV-6) is classified into variants A (HHV-6A) and B (HHV-6B) on the basis of distinct genetic, immunological, and biological characteristics. The HHV-6 genome contains open reading frames (ORFs) U1-U100 and flanking 8–9 kb terminal direct repeats (DRs) at either end.
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Publisher Summary Human herpesvirus-6 (HHV-6) is classified into variants A (HHV-6A) and B (HHV-6B) on the basis of distinct genetic, immunological, and biological characteristics. The HHV-6 genome contains open reading frames (ORFs) U1-U100 and flanking 8–9 kb terminal direct repeats (DRs) at either end.
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HHV-6 in Cardiovascular Pathology
2006Publisher Summary Among major clinical symptoms described for active human herpesvirus-6 (HHV-6) infection were palpitations, tachycardia, and arrhythmia as well as not-further specified ‘‘myocarditis'’. The cardiovascular system (CVS) may be a possible target for HHV-6 infections.
L. Maximilian Buja, L. Maximilian Buja
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HHV-6, 7 and their related diseases
Journal of Dermatological Science, 2000Human herpesvirus 6 (HHV-6) and human herpesvirus 7 (HHV-7) are relatively recently discovered beta-herpesvirus. They are prevalent in the human population. Primary infection of HHV-6 has been associated with exanthem subitum and febrile illness. Little information is known about the clinical characteristics of primary infection with HHV-7, although ...
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