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Decoding Multiple Keloids: A Gateway to Diagnosing Rubinstein-Taybi Syndrome.
Indian J DermatolJangid NC, Poriya R.
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Signs of Potential Androgen Excess Across the Lifespan in a US-based Digital Cohort Study.
J Clin Endocrinol MetabWolf AT +10 more
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International Journal of Dermatology, 1993
Hirsutism is the transformation of fine vellus hair to visible, thickened terminal hair under androgenic stimulus. The most common causes of hirsutism are familial, idiopathic or polycystic ovarian disease. Patient distress is the prime indication for therapy. Drug treatment should be continued for 12 months before assessing response. Spironolactone is
A K, Leung, W L, Robson
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Hirsutism is the transformation of fine vellus hair to visible, thickened terminal hair under androgenic stimulus. The most common causes of hirsutism are familial, idiopathic or polycystic ovarian disease. Patient distress is the prime indication for therapy. Drug treatment should be continued for 12 months before assessing response. Spironolactone is
A K, Leung, W L, Robson
openaire +4 more sources
Annals of Internal Medicine, 1987
Excess body hair, or hirsutism, is usually only a problem in women and can cause considerable psychological distress. The disorder is usually androgen mediated. Because androgens come only from the adrenal glands or gonads or by conversion in peripheral tissues of precursor steroids from these organs, the causes of hirsutism are found in these two ...
R S, Rittmaster, D L, Loriaux
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Excess body hair, or hirsutism, is usually only a problem in women and can cause considerable psychological distress. The disorder is usually androgen mediated. Because androgens come only from the adrenal glands or gonads or by conversion in peripheral tissues of precursor steroids from these organs, the causes of hirsutism are found in these two ...
R S, Rittmaster, D L, Loriaux
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Trends in Endocrinology & Metabolism, 1990
The clinical presentation and management of the hirsute woman is illustrated in cases of idiopathic hirsutism, polycystic ovarian disease, and nonclassical congenital adrenal hyperplasia. Therapeutic regimens manage hyperandrogenic signs, induce ovulation, and protect the endometrium.
J I, Mechanick, A, Dunaif
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The clinical presentation and management of the hirsute woman is illustrated in cases of idiopathic hirsutism, polycystic ovarian disease, and nonclassical congenital adrenal hyperplasia. Therapeutic regimens manage hyperandrogenic signs, induce ovulation, and protect the endometrium.
J I, Mechanick, A, Dunaif
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Primary Care: Clinics in Office Practice, 1977
Many women have some facial hair, and most patients who seek advice will be at the far end of the normal distribution curve for hair. In a second group there is a slight excessive production of androgens with associated hirsutism, either idiopathic or associated with polycystic ovarian disease.
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Many women have some facial hair, and most patients who seek advice will be at the far end of the normal distribution curve for hair. In a second group there is a slight excessive production of androgens with associated hirsutism, either idiopathic or associated with polycystic ovarian disease.
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Minerva pediatrica, 2007
--Hirsutism, defined as excessive growth of terminal hair in a male-type pattern, is prevalent in women and is most often a feature of androgen excess, associated with polycystic ovary syndrome (PCOS). Idiopathic hirsutism is the second most common cause.
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--Hirsutism, defined as excessive growth of terminal hair in a male-type pattern, is prevalent in women and is most often a feature of androgen excess, associated with polycystic ovary syndrome (PCOS). Idiopathic hirsutism is the second most common cause.
BONA G +9 more
openaire +4 more sources