Results 141 to 150 of about 29,485 (183)
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Evolutionary Dynamics of HTLV-I
Journal of Molecular Evolution, 2000Using mathematical models to describe the in vivo dynamics of HTLV-I infection, an explanation is offered for the slow rate of evolution of HTLV-I relative to HIV-1. In agreement with experimental findings, it is assumed that cell activation is required for successful replication in T helper cells and that HTLV-I induces a significant degree of ...
D, Wodarz, C R, Bangham
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Immunology & Cell Biology, 2007
Despite significant advances in our understanding of the immune response to persistent viruses like human T‐cell lymphotropic virus type I (HTLV‐I), many important questions remain unanswered. Mathematical modelling enables us to interpret and synthesise diverse experimental data in new ways and thus can contribute to our understanding. Here, we review
Becca, Asquith, Charles R M, Bangham
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Despite significant advances in our understanding of the immune response to persistent viruses like human T‐cell lymphotropic virus type I (HTLV‐I), many important questions remain unanswered. Mathematical modelling enables us to interpret and synthesise diverse experimental data in new ways and thus can contribute to our understanding. Here, we review
Becca, Asquith, Charles R M, Bangham
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The Epidemiology of HTLV-I in Europe
Journal of Acquired Immune Deficiency Syndromes and Human Retrovirology, 1996Although human T-lymphotropic virus type I (HTLV-I) infection in Europe is thought to be unusual except amongst people who have immigrated from countries where HTLV-I is endemic, the screening of blood donors has revealed a low seroprevalence across Europe, not only in donors originating from endemic areas but also in the indigenous population.
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Evidence of HTLV-I in thyroid tissue in an HTLV-I carrier with Hashimoto?s thyroiditis
Journal of Molecular Medicine, 1996Human T-lymphotropic virus type l (HTLV-I) protein and messenger RNA (mRNA) for HTLV-I were examined in thyroid tissues from two patients with Hashimoto's thyroiditis and serum anti-thyroid antibody. The virus envelope protein and signals for the mRNA were detected in many of the follicular epithelial cells of the thyroid tissue from one of the ...
H, Kawai +6 more
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The Journal of Immunology, 1987
Abstract Monoclonal antibodies against human T cell leukemia virus type I (HTLV-I) p24 and p19 were produced and employed in the in vivo expression of HTLV-I in some HTLV-I-related subjects by Western blot analysis. The antigenic determinants of these monoclonal antibodies were different from that of natural human anti-HTLV-I antibody ...
K, Ishibashi, S, Hanada, S, Hashimoto
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Abstract Monoclonal antibodies against human T cell leukemia virus type I (HTLV-I) p24 and p19 were produced and employed in the in vivo expression of HTLV-I in some HTLV-I-related subjects by Western blot analysis. The antigenic determinants of these monoclonal antibodies were different from that of natural human anti-HTLV-I antibody ...
K, Ishibashi, S, Hanada, S, Hashimoto
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Current Opinion in Immunology, 2000
A strong cytotoxic T lymphocyte response to HTLV-I protects against the associated inflammatory disease of the central nervous system, HAM/TSP (HTLV-I-associated myelopathy/tropical spastic paraparesis), by reducing the proviral load of HTLV-I; however, when the proviral load exceeds a threshold level, HTLV-I-specific cytotoxic T lymphocytes could ...
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A strong cytotoxic T lymphocyte response to HTLV-I protects against the associated inflammatory disease of the central nervous system, HAM/TSP (HTLV-I-associated myelopathy/tropical spastic paraparesis), by reducing the proviral load of HTLV-I; however, when the proviral load exceeds a threshold level, HTLV-I-specific cytotoxic T lymphocytes could ...
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The epidemiology of HTLV-I infection
Cancer Causes & Control, 1991It has been 10 years since the discovery of the human T-cell lymphotropic virus type I (HTLV-I), the first human retrovirus. During the past decade, significant progress has been made in understanding the transmission of the virus and defining its geographic distribution. It has been shown conclusively that HTLV-I is a causal factor in the induction of
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