Results 91 to 100 of about 8,334 (133)
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Pediatrics In Review, 1984
Neonatal hyperammonemia is a life-threatening complication of several conditions seen in the newborn infant (Table 1). Some of these disorders are potentially reversible; others are amenable to long-term therapy if early diagnosis and aggressive management in the neonatal period can prevent the catastrophic neurologic deterioration that accompanies ...
Steven M. Donn, Raul C. Banagale
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Neonatal hyperammonemia is a life-threatening complication of several conditions seen in the newborn infant (Table 1). Some of these disorders are potentially reversible; others are amenable to long-term therapy if early diagnosis and aggressive management in the neonatal period can prevent the catastrophic neurologic deterioration that accompanies ...
Steven M. Donn, Raul C. Banagale
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Hyperammonemia in Alzheimer's disease
American Journal of Psychiatry, 1985Postprandial blood ammonia levels were significantly higher in 22 patients with Alzheimer's disease than in 37 control subjects. In the Alzheimer group, fasting blood ammonia levels were significantly higher in patients whose EEGs showed triphasic waves than in patients without this change.
M, Fisman +5 more
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Severe Transient Neonatal Hyperammonemia
American Journal of Perinatology, 1986Severe transient hyperammonemia is a disorder of unknown etiology which can be successfully treated. This article describes two infants affected by this condition and reviews the pertinent literature. Forty-nine cases, including our own, are summarized.
G P, Giacoia, A, Padilla-Lugo
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Iatrogenic Hyperammonemia After Anorexia
Archives of Internal Medicine, 2010High-protein dietary supplements were started for 2 patients, who had a period of anorexia before hospital admission but no history of liver disease. Subsequent altered mental status with ataxia developed in both patients. After excluding other causes, hyperammonemia was noted, while liver function test results remained normal.
Emily, Welsh +2 more
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Hyperammonemia and Perinatal Asphyxia
Pediatrics, 1981In a recent article Goldberg et al1 presented data on 12 asphyxiated newborn infants with high ammonia levels concomitant with abnormal liver function tests. They pointed out that the liver dysfunction, probably secondary to the ischemic-hypoxic injury, might have contributed to ammonia elevation by decreasing urea biosynthesis.
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Tijdschrift voor kindergeneeskunde, 1983
Experience with severe neonatal hyperammonaemia in the Newborn Intensive Care Unit of the Leuven University Hospital is reported and compared with the literature data. Among eleven patients seven presented with the transient neonatal hyperammonaemia-syndrome and four with a urea cycle defect. Prompt recognition of the transient neonatal hyperammonaemia
J, Jaeken +4 more
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Experience with severe neonatal hyperammonaemia in the Newborn Intensive Care Unit of the Leuven University Hospital is reported and compared with the literature data. Among eleven patients seven presented with the transient neonatal hyperammonaemia-syndrome and four with a urea cycle defect. Prompt recognition of the transient neonatal hyperammonaemia
J, Jaeken +4 more
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Electroencephalographic changes and hyperammonemia
Electroencephalography and Clinical Neurophysiology, 1958Resume Sept patients souffrants de differentes maladies et dont l'etat de conscience variait entre un etat de vigilance normale jusqu'au coma profond ont montre des ondes ⪡ triphasiques ⪢ dans leur trace EEG. Tous les 7 malades avaient une perturbation commune, c'est-a-dire une elevation de l'ammonium sanguin. L'auteur tire les conclusions suivantes:
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Electroencephalographic changes in hyperammonemia
Electroencephalography and Clinical Neurophysiology, 1956Abstract 1. 1. Correlation of the electroencephalogram and the blood ammonia levels which have been obtained serially in two patients has been observed and the literature pertinent to this has been reviewed. 2. 2. The “critical” level of blood ammonia that corresponds with the first significant change in the electroencephalogram is 200 to 400
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Przeglad lekarski, 1999
Inherited hyperammonemia disorders are caused by specific enzymatic defects in the urea cycle or in metabolic pathways related to it. These disorders can be divided into the following groups: deficiencies of urea cycle enzymes, transport defects of dibasic amino acids, organic acidemias, defects in beta-oxidation of fatty acids, transient ...
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Inherited hyperammonemia disorders are caused by specific enzymatic defects in the urea cycle or in metabolic pathways related to it. These disorders can be divided into the following groups: deficiencies of urea cycle enzymes, transport defects of dibasic amino acids, organic acidemias, defects in beta-oxidation of fatty acids, transient ...
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