Results 301 to 310 of about 88,090 (336)
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Pathophysiology of Hypercalcemia
Endocrinology and Metabolism Clinics of North America, 2021Extracellular calcium is normally tightly regulated by parathyroid hormone (PTH), 1,25-dihydroxyvitamin D, as well as by calcium ion (Ca++) itself. Dysregulated PTH production leading to hypercalcemia occurs most commonly in sporadic primary hyperparathryoidism (PHPT) but may also result from select genetic mutations in familial disorders.
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The Journal of Clinical Endocrinology & Metabolism, 1962
ABSTRACT. Recently an adult patient with myxedema and hypercalcemia was observed to become normocalcemic when rendered euthyroid. This experience prompted a study of the effect of oral administration of calcium to hypothyroid patients and animals. Eleven patients with hypothyroidism, all of whom were normocalcemic, were given 30 ml of 40 % calcium ...
C. E. Lowe +2 more
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ABSTRACT. Recently an adult patient with myxedema and hypercalcemia was observed to become normocalcemic when rendered euthyroid. This experience prompted a study of the effect of oral administration of calcium to hypothyroid patients and animals. Eleven patients with hypothyroidism, all of whom were normocalcemic, were given 30 ml of 40 % calcium ...
C. E. Lowe +2 more
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Rhabdomyosarcoma and Hypercalcemia
Archives of Internal Medicine, 1989Hypercalcemia has not been reported as a complication of rhabdomyosarcoma in adults. We present the case of a 56-year-old man with pain in his right leg and hypercalcemia secondary to a large pelvic tumor. He had a rapidly progressive downhill course resulting in death due to tonsillar herniation.
Ray O. Lundy, Young S. Choi
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Postgraduate Medicine, 1979
Hypercalcemia is most commonly seen in normal infants as the result of normal rapid bone growth. The most common causative diseases are malignant disease and hyperparathyroidism. A variety of pharmacologic agents, especially vitamin D and its metabolites and thiazide diuretics, can elevate serum calcium levels.
Edward T. Zawada +2 more
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Hypercalcemia is most commonly seen in normal infants as the result of normal rapid bone growth. The most common causative diseases are malignant disease and hyperparathyroidism. A variety of pharmacologic agents, especially vitamin D and its metabolites and thiazide diuretics, can elevate serum calcium levels.
Edward T. Zawada +2 more
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Annals of Internal Medicine, 1969
Excerpt To the Editor:In the editorial note on the measurement of parathyroid hormone (PTH) (1) Dr.
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Excerpt To the Editor:In the editorial note on the measurement of parathyroid hormone (PTH) (1) Dr.
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Annals of Internal Medicine, 1970
Excerpt To the editor: Muggia and Heinemann, in their excellent article, "Hypercalcemia Associated with Neoplastic Disease,"Ann Intern Med73:281-290, 1970, state that sex hormone therapy of patient...
J, Horton +3 more
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Excerpt To the editor: Muggia and Heinemann, in their excellent article, "Hypercalcemia Associated with Neoplastic Disease,"Ann Intern Med73:281-290, 1970, state that sex hormone therapy of patient...
J, Horton +3 more
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The Electroencephalogram in Hypercalcemia
Archives of Neurology, 1967THE PURPOSE of this study was to determine what changes, if any, are produced in the electroencephalogram by a rise in the serum level of calcium.1 A low level of serum calcium is usually considered to be associated with a state of hyperexcitability of the central nervous system (CNS) with resultant tetany.
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Annals of Internal Medicine, 1978
Excerpt To the editor: Aldinger and Samaan in their article "Hypokalemia with Hypercalcemia" (Ann Intern Med87:571-573, 1977) demonstrate a higher prevalence of hypokalemia in the hypercalcemia of ...
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Excerpt To the editor: Aldinger and Samaan in their article "Hypokalemia with Hypercalcemia" (Ann Intern Med87:571-573, 1977) demonstrate a higher prevalence of hypokalemia in the hypercalcemia of ...
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Hypercalcemia and Hypermagnesemia
Veterinary Clinics of North America: Small Animal Practice, 1998Hypercalcemia and hypermagnesemia occur infrequently in small animal patients. Specific clinical signs usually do not accompany these electrolyte disorders, and their presence is often recognized only after reviewing serum chemistry profile results.
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