Results 111 to 120 of about 27,643 (258)

Fibroblast growth factor 23 and cardiovascular disease in patients with chronic kidney disease [PDF]

open access: yes, 2018
Chronic kidney disease (CKD) has been known to be associated with an increased risk of cardiovascular (CV) mortality and morbidity in Japan. Although traditional risk factors contribute to the development of CV disease in CKD patients, they cannot fully ...
NITTA Kosaku
core   +2 more sources

The effects of tenapanor on serum fibroblast growth factor 23 in patients receiving hemodialysis with hyperphosphatemia

open access: yesNephrology, Dialysis and Transplantation, 2018
Background Elevated serum fibroblast growth factor 23 (FGF23) is strongly associated with cardiovascular risk and mortality. Tenapanor, an inhibitor of gastrointestinal sodium/hydrogen exchanger isoform 3, decreased serum phosphate in a randomized ...
G. Block   +5 more
semanticscholar   +1 more source

Postoperative hyperphosphatemia significantly associates with adverse survival in colorectal cancer patients [PDF]

open access: yes, 2013
BACKGROUND: Hyperphosphatemia has been implicated in the development and treatment of various cancers. However, whether it can be used as a direct prognostic marker of colorectal cancer (CRC) has remained unexplored.
Guiles, Fran   +8 more
core   +1 more source

A Case of Neonatal Myositis Ossificans Related to Chronic Kidney Disease and Vitamin D Deficiency

open access: yesNephrology, Volume 30, Issue 11, November 2025.
ABSTRACT Myositis ossificans (MO) is currently defined as a self‐limiting ossifying lesion affecting any extra‐skeletal soft tissue type but mostly afflicting skeletal muscles. The pathogenetic mechanism of the disease is incompletely understood, especially in paediatric cases, and has been rarely described in neonates.
Dimitra Gialamprinou   +4 more
wiley   +1 more source

Clinical and pharmacoeconomic profile of lanthanum carbonate treatment of hyperphosphataemia in chronic renal dialysis patients

open access: yesFarmeconomia: Health Economics and Therapeutic Pathways, 2010
Hyperphosphatemia is recognized as a principal mineral disorder in chronic kidney disease (CKD) that leads to the development of secondary hyperparathyroidism.
Mario Eandi
doaj   +1 more source

Phosphate stimulates myotube atrophy through autophagy activation: evidence of hyperphosphatemia contributing to skeletal muscle wasting in chronic kidney disease

open access: yesBMC Nephrology, 2018
BackgroundAccelerated muscle atrophy is associated with a three-fold increase in mortality in chronic kidney disease (CKD) patients. It is suggested that hyperphosphatemia might contribute to muscle wasting, but the underlying mechanisms remain unclear ...
Yue-yue Zhang   +5 more
semanticscholar   +1 more source

Whitish Deposits in the Stomach and Duodenum

open access: yes
United European Gastroenterology Journal, EarlyView.
Kai‐yan Yang, Xu‐jie Zhou, Wei Xie
wiley   +1 more source

Diabetic Wound Repair: From Mechanism to Therapeutic Opportunities

open access: yesMedComm, Volume 6, Issue 10, October 2025.
Diabetic wound healing is compromised by chronic metabolic dysfunction that disrupts essential cellular processes. This review examines key predisposing factors, including neuropathy and vascular disease, characterizes dysfunction in critical cell types such as macrophages and fibroblasts, evaluates limitations of current models, compares established ...
Renyuan Wang   +5 more
wiley   +1 more source

Effects of Use of Proton Pump Inhibitors with Calcium Acetate on Serum Phosphorus Levels in Hemodialysis Patients [PDF]

open access: yesCaspian Journal of Internal Medicine, 2023
Background: The increase in serum phosphorus level is an independent risk factor for mortality in patients with chronic renal failure or undergoing dialysis due to end-stage renal disease.
Deniz Çekiç   +2 more
doaj  

Hipoparatiroidismo e pseudohipoparatiroidismo [PDF]

open access: yes, 2006
The principal function of the parathyroid hormone (PTH) is maintenance of calcium plasmatic levels, withdrawing the calcium from bone tissue, reabsorbing it from the glomerular filtrate, and indirectly increasing its intestinal absorption by stimulating ...
Borba, Victoria Zeghbi Cochenski   +4 more
core   +3 more sources

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