Results 221 to 230 of about 2,143,617 (291)

Incidentally detected double-chambered right ventricle in an adult. [PDF]

Radiol Case Rep
Saliba T, Rotzinger D, Fahrni G, Thoma P.   +3 more
europepmc   +1 more source

Lifestyle modification in atrial fibrillation: Mechanisms, phenotypes and ablation outcomes

Experimental Physiology, EarlyView.
Abstract Atrial fibrillation (AF) is the most prevalent sustained cardiac arrhythmia and is associated with significant morbidity, mortality and healthcare utilization. Catheter ablation is increasingly used as a rhythm‐control intervention for patients with symptomatic paroxysmal and persistent AF, yet recurrence rates remain suboptimal.
Konstantinos Grigoriou, Paschalis Karakasis, Panagiotis Theofilis, Konstantinos Pamporis, Panayotis K. Vlachakis, Anastasios Apostolos, Nikolaos Ktenopoulos, Barbara Fyntanidou, Efstratios Karagiannidis, Dimitrios Patoulias, Antonios P Antoniadis, Nikolaos Fragakis   +11 more
wiley   +1 more source

Epicardial Amyloidoma Mimicking Secondary Cardiac Lymphoma: A Sneeze Away From the Diagnosis. [PDF]

JACC Case Rep
Vella A, Disabato G, Roncoroni L, Lombardi M, Bedogni F.   +4 more
europepmc   +1 more source

Left Ventricular Hypertrophy [PDF]

, 2005
openaire   +1 more source

Mechanical pressure on endothelial cells mediates remote ischaemic preconditioning‐induced neuroprotection via miR‐126

Experimental Physiology, EarlyView.
Abstract Remote ischaemic preconditioning (RIPC) uses brief limb ischaemia to protect distant organs, but the mechanism of travel of the protective signal remains unclear. This study investigated whether endothelial cells (ECs) contribute to RIPC‐induced neuroprotection and the underlying mechanisms.
Xiaojie Wang, Lei Yan, Hongwei Zhu, Yifan Li, Qi Liu, Wei Li, Kerui Gong, Zhijun Zhao, Chunyang Zhang, Gang Fu, Guo Shao, Xunming Ji   +11 more
wiley   +1 more source

Genotype-Phenotype Associations in Pediatric Hypertrophic Cardiomyopathy: A Study From the Pediatric Cardiomyopathy Registry. [PDF]

JACC Heart Fail
Pahl E, Ware SM, Shi L, Colan SD, Everitt MD, Sleeper LA, Sridhar A, Schubert JA, Canter CE, Hsu DT, Webber SA, Kantor PF, Rossano JW, Chung WK, Lee TM, Towbin JA, Lal AK, Tariq M, Bhatnagar S, Dexheimer PJ, Aronow BJ, Martin LJ, Miller EM, Bansal N, Lipshultz SE.   +24 more
europepmc   +1 more source

Clearance of p16‐positive cardiac cells improves age‐related cardiac remodeling in mice

Experimental Physiology, EarlyView.
Abstract Senescent cells are characterized by expression of markers like p16 and secretion of profibrotic and proinflammatory factors. The role of cellular senescence in age‐related cardiac remodeling and dysfunction is incompletely understood. This study aimed to: (i) evaluate the effect of p16‐ positive cell clearance on cardiac function and ...
Mozhdeh Mehdizadeh, Martin Mackasey, Kimia Gharagozloo, Martin Aguilar, Patrice Naud, Allan Ochs, Nhung Vuong‐Robillard, Eric Thorin, Gerardo Ferbeyre, Jean‐Claude Tardif, Martin G. Sirois, Jean Francois Tanguay, Stanley Nattel   +12 more
wiley   +1 more source

Prevalence and determinants of asymmetric septal hypertrophy (ASH) in diseases of left ventricular hypertrophy (LVH). [PDF]

Eur Heart J Cardiovasc Imaging
Shiwani H, Augusto J, Topriceanu CC, Ioannou A, Rosmini S, Maestrini V, Hughes R, Knott K, Bhuva A, Dowsing B, Artico J, Pierce I, Treibel T, Howard J, Manisty C, Kellman P, Kozor R, Steeds R, Captur G, Mohiddin S, Lopes L, Fontana M, Moon J, Davies R.   +23 more
europepmc   +1 more source

A new approach to an old disease: Ageing, arrhythmogenic substrate and the role of risk‐factor modification in atrial fibrillation

Experimental Physiology, EarlyView.
Abstract Atrial fibrillation (AF) prevalence rises sharply with age due to two independent mechanisms. Ageing produces irreversible arrhythmogenic substrate changes through progressive fibrosis, cellular senescence and mitochondrial dysfunction, while modifiable risk factors such as obesity, hypertension, sleep apnoea, diabetes and alcohol use promote ...
Udit Thakur, Jonathan M. Kalman
wiley   +1 more source

TTN mutation-positive hypertrophic cardiomyopathy in congenitally corrected transposition of the great arteries: A rare coexistence. [PDF]

Ann Pediatr Cardiol
Chakraborty S, Dhulipudi B, Bhakru S, Koneti NR.   +3 more
europepmc   +1 more source