Results 81 to 90 of about 140,079 (313)

Animal models of chronic thromboembolic pulmonary hypertension

Animal Models and Experimental Medicine, EarlyView.
Current animal models of CTEPH. Created using BioRender.com. Abstract Chronic thromboembolic pulmonary hypertension (CTEPH) is a rare, yet life‐threatening disorder characterized by persistent pulmonary vascular obstruction and elevated pulmonary artery pressure, with progressive remodeling and subsequent right heart failure.
Yong‐Jian Zhu, Lin‐Chao Zhou, Xiao‐Xue Zhang, Xiao‐Yang Ren, Yi Yan, Shao‐Fei Liu   +5 more
wiley   +1 more source

Diagnostic value of R wave in aVL lead in left ventricular hypertrophy

Universidad Médica Pinareña, 2020
Introduction: left ventricular hypertrophy (LVH) is defined as the increase in the thickness of the wall or the interventricular septal portion (or both). This entity represents an important risk factor that is related to the increase in mortality.
Daniel Alejandro Denis-Piedra, Sheila Martínez-Gutiérrez, Albert Ramón Figueredo-Gilbert, Elia de la Caridad Rodríguez-Venegas   +3 more
doaj   +2 more sources

Equine models in translational medicine: A comparative approach to human health

Animal Models and Experimental Medicine, EarlyView.
This diagram summarizes and contrasts rodent and equine models, outlining their strengths, limitations, and applications. Horses offer naturally occurring diseases, genetic and physiological similarities to humans, and suitability for longitudinal and clinical‐scale studies.
Shayan Boozarjomehri Amnieh, Katarzyna Ropka‐Molik   +1 more
wiley   +1 more source

ASSESSMENT OF LEFT VENTRICULAR HYPERTROPHY IN PATIENTS WITH ESSENTIAL ARTERIAL HYPERTENSION IN A GENERAL PRACTICE

Zdravniški Vestnik, 2004
Background. The heart is a target organ in arterial hypertension. Echocardiography is the most valuable noninvasive method to assess the left ventricular hypertrophy.
Marija Petek Šter, Boris Krajačič
doaj  

Asociación de la hipertrofía ventricular izquierda con eventos cardiacos posteriores a intervencionismo coronario percutáneo. Association of left ventricular hypertrophy with cardiac events after percutaneous coronary intervention. [PDF]

Revista Cubana de Cardiología y Cirugía Cardiovascular, 2011
Introduction: Left ventricular hypertrophy is not included in the prognostic models of cardiacevents after percutaneous coronary intervention.Objective To determine the association of left ventricular hypertrophy with the presenceof cardiac events during
Luis R. Llerena Rojas, María B. Cabalé Vilariño, Amaury Flores Sánchez   +2 more
doaj  

Evaluation of cardiac histology and cell death markers during the progression of diabetes in a streptozotocin‐induced diabetes rat model

Animal Models and Experimental Medicine, EarlyView.
Daily insulin administration in streptozotocin‐induced diabetic rats prevented cardiac damage associated with diabetic cardiomyopathy, including hypertrophy, fibrosis, and expression of cell death markers, at 12 weeks. However, cyclophilin D, a marker of mitochondrial permeability transition pore‐opening‐associated cell death (necrosis), was observed ...
Tamara Sáez, Ivo Carrasco‐Wong, Juan Varas, Sebastián San Martín, Jaime A. Riquelme, Rienzi Díaz‐Navarro   +5 more
wiley   +1 more source

Diabetes, gender, and left ventricular structure in African-Americans: the atherosclerosis risk in communities study

Cardiovascular Ultrasound, 2006
Background Cardiovascular risk associated with diabetes may be partially attributed to left ventricular structural abnormalities. However, the relations between left ventricular structure and diabetes have not been extensively studied in African ...
Liebson Philip R, Benjamin Emelia J, Arnett Donna K, Duncan Bruce B, Foppa Murilo, Manolio Teri A, Skelton Thomas N   +6 more
doaj   +1 more source

Troponin release following endurance exercise: is inflammation the cause? a cardiovascular magnetic resonance study [PDF]

, 2010
Background: The aetiology and clinical significance of troponin release following endurance exercise is unclear but may be due to transient myocardial inflammation.
Wong Joyce, Whyte, G, Prasad, SK, Whyte, Greg, Smith Gillian, Annette Dahl, Smith, Gillian, Wage, R, Godfrey, R, Wilson, M, Gillian Smith, Francisco D Alpendurada, Sharma Sanjay, Prasad, Sanjay, O'Hanlon Rory, George Keith, Wage, Riccardo, Pennell, DJ, Pennell Dudley J, Roughton, Michael, Prasad Sanjay K, Roughton Michael, George, Keith, Rory O'Hanlon, Alpendurada Francisco D, Mat Wilson, Pennell, Dudley, Roughton, M, Sharma, S, Dahl, Annette, Greg Whyte, Sanjay K Prasad, Wilson Mat, Dave Oxborough, Alpendurada, Francisco, O'Hanlon, Rory, Keith George, Oxborough, Dave, Godfrey Richard, Wage Riccardo, Wong, Joyce, Dahl, A, Whyte Greg, Wilson, Mat, Smith, G, Joyce Wong, Richard Godfrey, Sanjay Sharma, Wong, J, Alpendurada, FD, Oxborough, D, Oxborough Dave, O'Hanlon, R, Dudley J Pennell, Riccardo Wage, Godfrey, Richard, Sharma, Sanjay, Michael Roughton, Dahl Annette, George, K   +59 more
core   +1 more source

Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response

Animal Models and Experimental Medicine, EarlyView.
In this study, we primarily simulated pathological myocardial remodeling induced by transverse aortic constriction surgery and found significant cardiac hypertrophy and fibrosis, characterized by deteriorated cardiac function. Oxidative stress response is considered as a pivotal pathological process, which contributes to inflammation and apoptosis of ...
Yijia Wang, Xing Feng, Shuhui Zhao, Yuchong Fu, Ao Zhang, Xiaofeng Shen, Bowen Yu, Yihao Wang, Jiahui Lin, Bing Zhang, Weiping Ji, Lianpin Wu, Xiaoling Guo   +12 more
wiley   +1 more source

Screening for Fabry Disease in Left Ventricular Hypertrophy: Documentation of a Novel Mutation

Arquivos Brasileiros de Cardiologia, 2015
Background: Fabry disease is a lysosomal storage disease caused by enzyme α-galactosidase A deficiency as a result of mutations in the GLA gene. Cardiac involvement is characterized by progressive left ventricular hypertrophy.
Ana Baptista, Pedro Magalhães, Sílvia Leão, Sofia Carvalho, Pedro Mateus, Ilídio Moreira   +5 more
doaj   +1 more source