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Recurrent Campylobacter Enteritis in Patients with Hypogammaglobulinemia: Review of the Literature
Recurrent Campylobacter enteritis is a well-recorded complication of primary hypogammaglobulinemia but has only rarely been reported with other types of immunodeficiency, and no cases have been reported after rituximab-associated secondary ...
Iris Najjar, Philip E Tarr
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Outcome of hypogammaglobulinemia in children: Immunoglobulin levels as predictors
We evaluated 131 children (M=88, F=43) with hypogammaglobulinemia Data was analyzed mainly for delineating predictor factors for outcome The distance from the lower limit of normal (-2SD) for any single measurement of immunoglobulins (Ig) was calculated ...
Ahmet Özen +2 more
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The outcome of patients with unclassified hypogammaglobulinemia in early childhood
Symptomatic hypogammaglobulinemia in childhood may be the initial finding of primary immunodeficiency (PID) or may be due to delay in maturation of immunoglobulin synthesis.
Necil Kutukculer, Nesrin Gülez
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The Genetics of Hypogammaglobulinemia
Current Allergy and Asthma Reports, 2004Etiologies for human hypogammaglobulinemias are diverse and include genetic and nongenetic causes. Although recent reviews focus on the complex genetics of common variable immunodeficiency, in this review, we survey different causes of hypogammaglobulinemias and discuss possible mechanisms.
Grimbacher, Bodo +2 more
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Hypogammaglobulinemia and steatorrhea
The American Journal of Digestive Diseases, 19651. A patient with acquired idiopathic hypogammaglobulinemia and steatorrhea is described. Jejunal and ileal biopsies were histologically normal.
C F, McCarthy, W I, Austad, A E, Read
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Heterogeneity of hypogammaglobulinemia
American Journal of Hematology, 1980AbstractIn 1952, Ogden Bruton described the first patient with recurrent infections and hypogammaglobulinemia [1]. For many years thereafter, hypogammaglobulinemia (or agammaglobulinemia) was believed to result from a deficiency (or absence) of the antibody‐producing cells, or “B” cell line of the lymphoid system.
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DIAGNOSIS OF HYPOGAMMAGLOBULINEMIA
JAMA, 1962Prompt, accurate diagnosis of gamma globulin deficiency as a cause of unusual susceptibility to infection is extremely important for several reasons: first, replacement therapy with pooled normal gamma globulin is effective in the prevention of serious infections if initiated before structural damage has occurred; second, supplies of gamma globulin ...
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Pediatrics, 1960
An unusual patient, characterized by recurrent infections, hepatosplenomegaly, low levels of gamma-globulin in the serum (400 mg/100 ml) and lymphocytosis, is described. The patient had pneumonia due to Nocardia asteroides followed by chronic Salmonella newport infection of 16 months' duration, kept in check by continuous antibiotic ...
Thomas F. Dolan +2 more
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An unusual patient, characterized by recurrent infections, hepatosplenomegaly, low levels of gamma-globulin in the serum (400 mg/100 ml) and lymphocytosis, is described. The patient had pneumonia due to Nocardia asteroides followed by chronic Salmonella newport infection of 16 months' duration, kept in check by continuous antibiotic ...
Thomas F. Dolan +2 more
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Hypogammaglobulinemia and Pernicious Anemia
Southern Medical Journal, 1987We have described two patients with pernicious anemia in whom incidental hypogammaglobulinemia was discovered. According to our review of 41 cases of this association reported in the literature, our patients are similar to those previously reported in their young age, the evidence for antral involvement with the atrophic gastritis, and the absence in ...
P E, Wright, D A, Sears
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An atypical case of hypogammaglobulinemia
Annals of Allergy, Asthma & Immunology, 2000LABORATORY AND DIAGNOSTIC STUDIES The complete blood count showed a white count of 4500/mm3. He had a low hematocrit of 33%. The platelet count was normal. The renal profile, liver enzymes, and bilirubin were within normal limits. The chest radiograph revealed a thymic shadow. Pertinent details of the immunologic evaluation are stated in Tables 1, 2, 3.
A, Mansur +3 more
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