Results 121 to 130 of about 21,660 (161)
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Hypophosphatemia and growth

Pediatric Nephrology, 2012
Over the last decade the discovery of fibroblast growth factor 23 (FGF23) and the progressive and ongoing clarification of its role in phosphate and mineral metabolism have led to expansion of the diagnostic spectrum of primary hypophosphatemic syndromes. This article focuses on the impairment of growth in these syndromes.
Rocío Fuente   +5 more
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Hypophosphatemia in Hospitalized Patients

JAMA: The Journal of the American Medical Association, 1979
Hypophosphatemia is common in hospitalized patients and occurs under a variety of circumstances other than parathyroid hormone excess. Charts of 100 inpatients with hypophosphatemia were reviewed and the patients divided into five groups on the basis of serum phosphate level: 18, 2.1 to 2.4 mg/dL; 49, 1.6 to 2.0 mg/dL; 20, 1.1 to 1.5 mg/dL; 12, 0.6 to ...
Mohamed A. Elrazak, David Juan
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Hypophosphatemia

Drug Intelligence & Clinical Pharmacy, 1984
Hypophosphatemia, defined as serum phosphate levels <2.5 mg%, is a relatively common disorder that can affect virtually every organ system. Phosphate deficiency can result from decreases in phosphate intake or absorption, increased loss from renal and nonrenal pathways, and transcellular phosphate shifts.
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The pathophysiology of hypophosphatemia

Best Practice & Research Clinical Endocrinology & Metabolism
After identification of fibroblast growth factor (FGF) 23 as the pivotal regulator of chronic serum inorganic phosphate (Pi) levels, the etiology of disorders causing hypophosphatemic rickets/osteomalacia has been clarified, and measurement of intact FGF23 serves as a potent tool for differential diagnosis of chronic hypophosphatemia.
Nobuaki, Ito   +2 more
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Acquired Hypophosphatemia

Endocrinology and Metabolism Clinics of North America, 1993
This article discusses the regulation of serum phosphorus under normal conditions, focusing on the pathophysiology of acquired hypophosphatemia and its clinical manifestation. In addition, the clinical settings and conditions in which hypophosphatemia is likely to cause severe morbidity are emphasized.
S F, Hodgson, D L, Hurley
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Hypophosphatemia in the Alcoholic

Archives of Internal Medicine, 1980
Elsewhere in this issue (see p 673), Ryback and his associates confirm the common occurrence 1 of hypophosphatemia in hospitalized alcoholics. It is important to recognize this finding since either hypophosphatemia or phosphorus deficiency may play an important role in the morbidity of the long-term alcoholic. The ubiquitous, poorly nourished alcoholic
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Treatment of Refractory Hypophosphatemia

Southern Medical Journal, 1982
Profound hypophosphatemia developed in a patient with chronic alcoholism. Multiple causative factors were identified and were thought to account for the initial failure of phosphorus repletion. High-dose intravenous phosphorus was effective in restoring serum phosphorus to normal with no observable adverse effects.
R Whang, D L Andress, Jerry B. Vannatta
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Treatment of hypophosphatemia

Journal of the American College of Surgeons, 2004
hank you for your comments. You highlight one of the oints we tried to make in our study of pathology reorting of breast specimens: Good pathology reporting s the result of a collaborative effort between the surgeon nd the pathologist. You are also correct that failure to anage the breast specimen appropriately can result in dditional operations that ...
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