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Admission hyperphosphatemia as a predictor of severity and mortality in acute pancreatitis: A 1000-patient cohort study. [PDF]
World J Gastrointest SurgÖzden Y, Buyukberber NM.
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AJN, American Journal of Nursing, 1985
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Vincent, Jean Louis, Goldstein, Jacques
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Vincent, Jean Louis, Goldstein, Jacques
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The pathophysiology of hypophosphatemia
Best Practice and Research in Clinical Endocrinology and MetabolismAfter identification of fibroblast growth factor (FGF) 23 as the pivotal regulator of chronic serum inorganic phosphate (Pi) levels, the etiology of disorders causing hypophosphatemic rickets/osteomalacia has been clarified, and measurement of intact FGF23 serves as a potent tool for differential diagnosis of chronic hypophosphatemia.
N Itô, Naoko Hidaka, Hajime Kato
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Pediatric Nephrology, 2012
Over the last decade the discovery of fibroblast growth factor 23 (FGF23) and the progressive and ongoing clarification of its role in phosphate and mineral metabolism have led to expansion of the diagnostic spectrum of primary hypophosphatemic syndromes. This article focuses on the impairment of growth in these syndromes.
Fernando, Santos +5 more
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Over the last decade the discovery of fibroblast growth factor 23 (FGF23) and the progressive and ongoing clarification of its role in phosphate and mineral metabolism have led to expansion of the diagnostic spectrum of primary hypophosphatemic syndromes. This article focuses on the impairment of growth in these syndromes.
Fernando, Santos +5 more
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Endocrinology and Metabolism Clinics of North America, 1993
This article discusses the regulation of serum phosphorus under normal conditions, focusing on the pathophysiology of acquired hypophosphatemia and its clinical manifestation. In addition, the clinical settings and conditions in which hypophosphatemia is likely to cause severe morbidity are emphasized.
S F, Hodgson, D L, Hurley
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This article discusses the regulation of serum phosphorus under normal conditions, focusing on the pathophysiology of acquired hypophosphatemia and its clinical manifestation. In addition, the clinical settings and conditions in which hypophosphatemia is likely to cause severe morbidity are emphasized.
S F, Hodgson, D L, Hurley
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