Results 181 to 190 of about 24,867 (235)
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Endocrinology and Metabolism Clinics of North America, 1993
This article discusses the regulation of serum phosphorus under normal conditions, focusing on the pathophysiology of acquired hypophosphatemia and its clinical manifestation. In addition, the clinical settings and conditions in which hypophosphatemia is likely to cause severe morbidity are emphasized.
S F, Hodgson, D L, Hurley
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This article discusses the regulation of serum phosphorus under normal conditions, focusing on the pathophysiology of acquired hypophosphatemia and its clinical manifestation. In addition, the clinical settings and conditions in which hypophosphatemia is likely to cause severe morbidity are emphasized.
S F, Hodgson, D L, Hurley
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Drug Intelligence & Clinical Pharmacy, 1984
Hypophosphatemia, defined as serum phosphate levels <2.5 mg%, is a relatively common disorder that can affect virtually every organ system. Phosphate deficiency can result from decreases in phosphate intake or absorption, increased loss from renal and nonrenal pathways, and transcellular phosphate shifts.
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Hypophosphatemia, defined as serum phosphate levels <2.5 mg%, is a relatively common disorder that can affect virtually every organ system. Phosphate deficiency can result from decreases in phosphate intake or absorption, increased loss from renal and nonrenal pathways, and transcellular phosphate shifts.
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Hypophosphatemia-induced Cardiomyopathy
The American Journal of the Medical Sciences, 2016Relatively few studies have been conducted to evaluate the effect of hypophosphatemia on cardiac function. The goal of this review was to determine whether there is an association between hypophosphatemia and cardiac function and to increase awareness of hypophosphatemia-induced cardiomyopathy as a new clinical entity and a reversible cause of heart ...
Nobuhiro, Ariyoshi +4 more
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Pediatric Nephrology, 2012
Over the last decade the discovery of fibroblast growth factor 23 (FGF23) and the progressive and ongoing clarification of its role in phosphate and mineral metabolism have led to expansion of the diagnostic spectrum of primary hypophosphatemic syndromes. This article focuses on the impairment of growth in these syndromes.
Fernando, Santos +5 more
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Over the last decade the discovery of fibroblast growth factor 23 (FGF23) and the progressive and ongoing clarification of its role in phosphate and mineral metabolism have led to expansion of the diagnostic spectrum of primary hypophosphatemic syndromes. This article focuses on the impairment of growth in these syndromes.
Fernando, Santos +5 more
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Archives of Internal Medicine, 1963
The etiology of rachitic bone disease associated with hypophosphatemia, which requires large doses of vitamin D for treatment, has been a subject of dispute. Albright, Butler, and Bloomberg 1 have attributed the syndrome to parathyroid hyperplasia secondary to calcium malabsorption, whereas Fanconi and Girardet 2 have proposed an intrinsic and primary ...
J E, WHITE +3 more
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The etiology of rachitic bone disease associated with hypophosphatemia, which requires large doses of vitamin D for treatment, has been a subject of dispute. Albright, Butler, and Bloomberg 1 have attributed the syndrome to parathyroid hyperplasia secondary to calcium malabsorption, whereas Fanconi and Girardet 2 have proposed an intrinsic and primary ...
J E, WHITE +3 more
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The pathophysiology of hypophosphatemia
Best Practice & Research Clinical Endocrinology & MetabolismAfter identification of fibroblast growth factor (FGF) 23 as the pivotal regulator of chronic serum inorganic phosphate (Pi) levels, the etiology of disorders causing hypophosphatemic rickets/osteomalacia has been clarified, and measurement of intact FGF23 serves as a potent tool for differential diagnosis of chronic hypophosphatemia.
Nobuaki, Ito +2 more
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