Results 61 to 70 of about 3,545 (197)
Incision at hypoxanthine residues in DNA by a mammalian homologue of the Escherichia coli antimutator enzyme endonuclease V [PDF]
Ardis Moe
openalex +1 more source
Fried potato consumption is increasingly associated with metabolic disorders, yet preventive strategies remain poorly understood. In a 13‐week mouse model, long‐term potato chip intake induced significant glycolipid metabolic disturbances and tissue damage, which were ameliorated by fish oil supplementation through modulation of inflammation, hepatic ...
Xuzhi Wan +10 more
wiley +1 more source
Anodic Voltammetry and Its Analytical Application to the Detection and Simultaneous Determination of Hypoxanthine, Xanthine, and Uric Acid [PDF]
Toshio Yao +3 more
openalex +1 more source
Lys–Leu MRPs heat‐treated for 2 h exhibited high antioxidant activity in vitro. A treatment with Lys–Leu MRPs extends the lifespan and health span of C. elegans by regulating ROS levels through their inherent antioxidant activity and the IIS pathway.
Yaqi Jia +4 more
wiley +1 more source
High-risk genotypes for type 1 diabetes are associated with the imbalance of gut microbiome and serum metabolites. [PDF]
Yue T +10 more
europepmc +1 more source
Xanthine Accumulation during Hypoxanthine Oxidation by Milk Xanthine Oxidase [PDF]
Maria Jeżewska
openalex +1 more source
Peanut sprout extract (PSE) significantly attenuated skeletal muscle atrophy induced by a high‐fat, high‐sucrose diet and dexamethasone, improving muscle strength and reducing triglyceride accumulation and inflammation. It enhanced mitochondrial function by upregulating TFAM and OXPHOS proteins and partially restoring PGC1α expression.
Sang‐Mi Jo +4 more
wiley +1 more source
Abnormalities of neural stem cells in Lesch-Nyhan disease. [PDF]
Dinasarapu AR +4 more
europepmc +1 more source
Hypoxanthine is a pharmacodynamic marker of ischemic brain edema modified by glibenclamide [PDF]
Hannah Irvine +8 more
openalex +1 more source
Treatable Neonatal Molybdenum Cofactor Deficiency: Rapid Demise Despite Rapid Biochemical Diagnosis
ABSTRACT Molybdenum cofactor deficiency (MoCD) is an inborn error of metabolism included in the differential for refractory neonatal seizures. The prognosis is guarded, with a median reported age of death between 2.4 and 3.0 years. Mortality is primarily due to seizures and lower respiratory tract infections.
Molly M. Crenshaw +11 more
wiley +1 more source

