Results 1 to 10 of about 46,054 (219)

Restenosis of Endovascular Stents From Stent Compression

Journal of the American College of Cardiology, 1997
We sought to determine the basis for restenosis within superficial femoral arteries (SFAs) and hemodialysis conduits treated with balloon-expandable stents.Use of stents within coronary and peripheral vessels continues to increase exponentially. The mechanism of restenosis within stents placed at various vascular sites is not well understood.
Kenneth Rosenfield, Robert M. Schainfeld, Ann Pieczek, Laura Haley, Jeffrey M. Isner   +4 more
openalex   +3 more sources

Current Management of In-Stent Restenosis. [PDF]

J Clin Med
In-stent restenosis (ISR) remains the primary cause of target lesion failure following percutaneous coronary intervention (PCI), resulting in 10-year incidences of target lesion revascularization at a rate of approximately 20%. The treatment of ISR is challenging due to its inherent propensity for recurrence and varying susceptibility to available ...
Giacoppo D, Mazzone PM, Capodanno D.
europepmc   +3 more sources

In-Stent Restenosis [PDF]

Cardiovascular Therapeutics, 2010
The introduction of coronary stents marked a major turning point in the practice of interventional cardiology. Whereas the efficacy of balloon angioplasty was challenged both by immediate mechanical complications and by a high incidence of restenosis, coronary stents offered cardiologists a means by which to not only augment immediate procedural ...
Michael S. Kim, Larry S. Dean
openaire   +3 more sources

Predictors of In-Stent Restenosis [PDF]

Angiology, 2018
Abstract Not ...
Mehmet Kadri Akboga, Samet Yilmaz
openaire   +4 more sources

Calcified in-stent restenosis in a venous stent [PDF]

Journal of Vascular Surgery Cases, 2015
Stenting of the iliac veins has been an established treatment for improving venous runoff from the legs after thrombolysis of iliofemoral deep venous thrombosis for more than a decade, yet little is known about the long-term fate of stents in the central veins. We describe a case of heavily calcified in-stent restenosis in a 10-year-old venous stent as
Schmidt, Jens-Ove, Christensen, Johnny, Houlind, Kim Christian   +2 more
openaire   +5 more sources

Implantation of paclitaxel-eluting stents in saphenous vein grafts: clinical and angiographic follow-up results from a multicentre study. [PDF]

, 2006
Objective: To define the clinical and angiographic follow-up results after implantation of paclitaxel-eluting stents (PESs) in stenotic saphenous vein grafts (SVGs). Design: Prospective multicentre study. Comparison with a control group.
Blindt, Ruediger, Boeckstegers, Peter, Heitzer, Thomas, Hoffmann, Rainer, Köster, Ralf, Pohl, Tilmann   +5 more
core   +2 more sources

Inhibition of in-stent stenosis by oral administration of bindarit in porcine coronary arteries [PDF]

, 2011
<p><b>Objective:</b> We have previously demonstrated that bindarit, a selective inhibitor of monocyte chemotactic proteins (MCPs), is effective in reducing neointimal formation in rodent models of vascular injury by reducing smooth ...
Baker, A.H., Biondi, G., Colombo, A., Di Lauro, M.V., Gordon, P., Grassia, G., Guglielmotti, A., Ialenti, A., Kennedy, S., Maddaluno, M., Maffia, P.   +10 more
core   +1 more source

Dealing with in-stent restenosis [PDF]

Heart, 1998
Stents have been shown to reduce the incidence of restenosis; however, they are not a cure. In two trials the angiographic restenosis rate was reported to be between 20% and 30% (in the Benestent II trial the unpublished quoted restenosis rate was 15%). These trials were based on stenting short, de novo lesions, in native coronary arteries.
J Baron, Anthony H. Gershlick
openaire   +3 more sources

A polymer coated cicaprost-eluting stent increases neointima formation and impairs vessel function in the rabbit iliac artery [PDF]

, 2016
Drug-eluting stents have been successful in reducing in-stent restenosis but are not suitable for all lesion types and have been implicated in causing late stent thrombosis due to incomplete regeneration of the endothelial cell layer.
Jones, Robert L., Kennedy, Simon, McCormick, Christopher, Mullen, Alexander B., Wadsworth, Roger M.   +4 more
core   +2 more sources

Pathogen burden, inflammation, proliferation and apoptosis in human in-stent restenosis - Tissue characteristics compared to primary atherosclerosis [PDF]

, 2004
Pathogenic events leading to in-stent restenosis (ISR) are still incompletely understood. Among others, inflammation, immune reactions, deregulated cell death and growth have been suggested. Therefore, atherectomy probes from 21 patients with symptomatic
Abdelmegui AE, Alexander Jabs, Bauriedel G, Bauriedel G, Berndt Lüderitz, Carter AJ, Chiu B, Dirk Skowasch, Epstein SE, Farb A, Gerhard Bauriedel, Kearney M, Kol A, Komatsu R, Mayr M, Moreno PR, Neumann FJ, Nicolas Wernert, O'Brien ER, Pickering JG, René Andrié, Sabine Dinkelbach, Simonton CA, Thomas M. Schiele, Zohlnhofer D   +24 more
core   +1 more source