Results 301 to 310 of about 635,488 (322)

Mediators of Inflammation and Fibrosis

Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis, 2007
During peritoneal dialysis, peritoneal cells are repeatedly exposed to a non-physiologic hypertonic environment with high glucose content and low pH. Current sterile dialysis solutions cause inflammation in the submesothelial compact zone, leading to fibrosis, angiogenesis, and, eventually, ultrafiltration failure.
Kar Neng, Lai, Sydney C W, Tang, Joseph C K, Leung   +2 more
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Mediators of Inflammation and Regeneration

Advances in Dental Research, 2011
Characterization of the molecular response under caries lesions requires a robust and reliable transcript isolation system, and analysis of data indicated that collection of extracted teeth in either liquid nitrogen/RNA-stabilizing solution facilitated this.
P R, Cooper, J L, McLachlan, S, Simon, L W, Graham, A J, Smith   +4 more
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Mediators of Injury and Inflammation

World Journal of Surgery, 1996
AbstractMediators play a key role in the development of systemic inflammatory response syndrome (SIRS), multiple organ dysfunction syndrome, and multiple organ failure of vital organs. In this short review, we update our knowledge on these mediator networks.
G, Schlag, H, Redl
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Platelets as Mediators of Inflammation

Hematology/Oncology Clinics of North America, 2007
An expanding body of evidence continues to build on the central role of inflammation in the progression and clinical manifestations of atherosclerosis. Platelets, long thought to play only a reactionary role at the time of endothelial disruption, are now recognized as important mediators of the inflammatory process.
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Cytokine mediators of renal inflammation

Current Opinion in Nephrology and Hypertension, 1994
This review focuses on the role of two cytokines, platelet-derived growth factor and transforming growth factor-beta, in glomerular injury. The most recent in vitro and in vivo evidence for the role of platelet-derived growth factor and transforming growth factor-beta in glomerular hypercellularity and matrix expansion is summarized.
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Mediators of Inflammation

Annual Review of Immunology, 1983
G L, Larsen, P M, Henson
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Mediators of Anaphylaxis and Inflammation

Annual Review of Microbiology, 1982
INTRODUCTION .. . . .. .. .. . .. .. .. .. .... .. .. . .. .... . .. .. . ....... .. .. ... .. . .... . .. .. . .. . .. . .. .... . .. . .. .... ... ... . . .. ..... .. 371 Release of Mediators of Anaphylaxis ... ..... .. ....... . .. .. ..... 372 Substances Considered in this Review 373 MAST CELL-DERIVED PREFORMED MEDIATORS .. . . . .. . .. ..
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Mediators of Inflammation

1974
1. The Mediators of Inflammation.- 2. Mechanisms Common to Several Mediator Systems.- 2.1. Cellular Release.- 2.2. Fluid-Phase Activation.- 2.3. Bypass Mechanisms.- 2.4. Extracellular Control Loops.- 2.5. Intracellular Controls.- 3. Centrality of Phagocytes.- 4. References.- 1 Mechanisms of Mediator Release from Inflammatory cells.- 1. Introduction.- 2.
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Biological Mediators of Acute Inflammation

AACN Clinical Issues: Advanced Practice in Acute and Critical Care, 2004
Inflammation may be defined as the normal response of living tissue to injury or infection. It is important to emphasize two components of this definition. First, that inflammation is a normal response and, as such, is expected to occur when tissue is damaged.
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Bacteria-Mediated Acute Lung Inflammation

2013
Mouse models of acute lung inflammation are critical for understanding the role of the innate immune response to pathogen associated molecular patterns, bacteria, and sepsis in humans. Bacterial infections in the lung elicit a range of immune reactions, depending on the pathogen, the level of exposure and the effectiveness of the host response.
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