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Mechanisms of Inflammatory Pain
JCR: Journal of Clinical Rheumatology, 2004The study and treatment of clinical pain has historically identified particular pain syndromes and linked their etiology with disease factors. Missing in this approach is consideration of the mechanisms accounting for the pain that is experienced by the patient.
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Aceclofenac in the management of inflammatory pain
Expert Opinion on Pharmacotherapy, 2004Aceclofenac (Almirall Prodesfarma SA) is an oral NSAID that is effective in the treatment of painful inflammatory diseases and has been used to treat > 75 million patients worldwide. It has proved as effective as diclofenac, naproxen and piroxicam in patients with osteoarthritis, diclofenac, ketorolac, tenoxicam and indomethacin in patients with ...
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NGF as a mediator of inflammatory pain
Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences, 1996Abstract The chapter reviews some of recent evidence which suggests that one neurotrophin, nerve growth factor (NGF), is a peripherally produced mediator of some persistent pain states, notably those associated with inflammation. The evidence for this proposal is as follows. 1.
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2016
Inflammatory back pain (IBP) refers to a collection of symptoms that may help identify patients with possible inflammatory spinal disease. A number of criteria sets have been proposed for IBP, which share common features such as: onset of symptoms aged <40 years, alternating buttock pain, improvement with exercise, worsening with rest, gradual onset,
Stefan Siebert +2 more
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Inflammatory back pain (IBP) refers to a collection of symptoms that may help identify patients with possible inflammatory spinal disease. A number of criteria sets have been proposed for IBP, which share common features such as: onset of symptoms aged <40 years, alternating buttock pain, improvement with exercise, worsening with rest, gradual onset,
Stefan Siebert +2 more
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Electrophysiological Mechanisms in Inflammatory Pain
1991Inflammatory pain is mediated from the periphery by a specialized subgroup of nerve fibers, the nociceptors. The nociceptive nerve endings undergo sensitization and excitation when stimulated with inflammatory agents. It will be shown that a multitude of substances cooperates in this process.
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Pain and Inflammatory Mediators
1978Pain and hyperalgesia are common features of the inflammatory process. Some pain arises as an immediate sensation after tissue injury, due to direct stimulation of sensory nerve endings. Another component arises with the inflammation which ensues after injury; the pain which then develops is a combination of mechanical and chemical stimulation due to ...
S. Moncada, S. H. Ferreira, J. R. Vane
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Macrophages transfer mitochondria to sensory neurons to resolve inflammatory pain
Neuron, 2022Michiel Van Der Vlist +2 more
exaly
Proteases and Inflammatory Pain
2011Inflammation mediators are known to signal neurons and provoke pain. In this context, proteases can signal sensory neurons at the site of inflammation and modulate pain transmission. In this chapter, we will perform an overview of the multiple mechanisms whereby proteases can signal nerve endings.
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Nabilone in inflammatory pain: to be or not to be
Clinical and Experimental Pharmacology and Physiology, 2012openaire +2 more sources
1977
The existence of an ongoing inflammatory process is generally indicated by pain or fever and these symptoms are also the first to be relieved by nonsteroidal anti-inflammatory therapy. This is a brief review of the participation of prostaglandins in the genesis of these symptoms.
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The existence of an ongoing inflammatory process is generally indicated by pain or fever and these symptoms are also the first to be relieved by nonsteroidal anti-inflammatory therapy. This is a brief review of the participation of prostaglandins in the genesis of these symptoms.
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