Results 81 to 90 of about 131,926 (308)

Imperative role of adaptor proteins in macrophage toll-like receptor signaling pathways

open access: yesFuture Science OA
Macrophages are integral part of the body's defense against pathogens and serve as vital regulators of inflammation. Adaptor molecules, featuring diverse domains, intricately orchestrate the recruitment and transmission of inflammatory responses through ...
Aurelia Rughetti   +10 more
doaj   +1 more source

NECESSARY AND SUFFICIENT CONDITIONS OF (α, β)-ACCESSIBILITY OF DOMAIN IN NONSMOOTH CASE

open access: yesПроблемы анализа, 2015
In [1], [2] (α, β)-accessible domains in C were defined and investigated, and the criterion of (α, β)-accessibility in a smooth case was obtained. (α, β)-accessible domains are starlike, γ-accessible (γ = min{α, β}), and satisfy the so-called “cone ...
K. F. Amozova
doaj   +1 more source

Adenosine‐to‐inosine editing of miR‐200b‐3p is associated with the progression of high‐grade serous ovarian cancer

open access: yesMolecular Oncology, EarlyView.
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira   +14 more
wiley   +1 more source

Evolutionary equation of inertial waves in 3-D multiply connected domain with Dirichlet boundary condition

open access: yesInternational Journal of Mathematics and Mathematical Sciences, 2001
We study initial-boundary value problem for an equation of composite type in 3-D multiply connected domain. This equation governs nonsteady inertial waves in rotating fluids.
Pavel A. Krutitskii
doaj   +1 more source

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

open access: yesMolecular Oncology, EarlyView.
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian   +10 more
wiley   +1 more source

A novel approach to nonlinear fractional volterra integral equations

open access: yesActa Polytechnica
Nonlinear Fractional Volterra integral equations (FVIEs) of the first kind present challenges due to their intricate nature, combining fractional calculus and integral equations.
Mohammed Abdulshareef Hussein   +1 more
doaj   +1 more source

Integral domains of finite t-character

open access: yesJournal of Algebra, 2013
Let \(D\) be an integral domain. Then we say that \(D\) has \textit{finite character} if any nonzero element of \(D\) belongs only to finitely many maximal ideals of \(D\). The finite character of a domain is strictly connected to the multiplicative structure of the ideals of the domain.
Anderson, D. D.   +2 more
openaire   +1 more source

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

Factorization in integral domains, II

open access: yesJournal of Algebra, 1992
Les AA. continuent l'étude des propriétés des anneaux intègres, qui sont plus faibles que les propriétés de anneaux factoriels, commencé dans la première partie du papier [cf. J. Pure Appl. Algebra 69, No. 1, 1-19 (1990; Zbl 0727.13007)]. Dans cette deuxième partie sont étudiés les comportements des propriétés pour les extensions d'anneaux intègres ...
Anderson, D.D   +2 more
openaire   +1 more source

Emerging role of ARHGAP29 in melanoma cell phenotype switching

open access: yesMolecular Oncology, EarlyView.
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster   +3 more
wiley   +1 more source

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