Results 181 to 190 of about 27,999 (220)

Spontaneous Intracranial Hypotension Without Intracranial Hypotension

Journal of Neuro-Ophthalmology, 2011
A 52-year-old man with a family history of multiple aneurysms presented with the gradual onset of generalized headaches and bilateral sixth nerve palsies. Following intravenous contrast, MRI revealed diffuse pachymeningeal enhancement consistent with spontaneous intracranial hypotension (SIH).
Tasneem, Peeraully, Michael L, Rosenberg
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Intracranial hypotension

Neurology, 1996
Intracranial hypotension (IH) is present when cerebrospinal fluid (CSF) pressure is 60mm H2O or lower and there has been no previous dural puncture. IH is more common in women than in men (3:1). Orthostatic headache is the cardinal symptom. Visual, auditory, and other symptoms occur.
M S, Hochman, T P, Naidich
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Intracranial Hypotension

Journal of Neurosurgery, 2008
The presentation of spontaneous intracranial hypotension (SIH) can be associated with various clinical and neuroimaging features that may impede a rapid diagnosis of this entity. The authors report the case of a patient who presented with bilateral third cranial nerve palsies and bilateral subdural hematomas.
Albayram, Mehmet Sait, Onur Ulu, Mustafa Onur, İşler, Cihan   +2 more
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Spontaneous intracranial hypotension

American Journal of Ophthalmology, 1999
To describe a patient with classic presentation of spontaneous intracranial hypotension and subsequent improvement with targeted epidural blood patch.Report of one case and review of the literature.Examination of cerebrospinal fluid after lumbar puncture disclosed a reduced opening pressure, an increased level of protein, and lymphocytic pleocytosis ...
R S, Apte, W, Bartek, A, Mello, A, Haq
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Spontaneous Intracranial Hypotension

Archives of Neurology, 2002
Spontaneous intracranial hypotension (SIH) is an increasingly recognized syndrome. Postural headache with typical findings on magnetic resonance imaging (MRI) are the key to diagnosis. Delay in diagnosing this condition may subject patients to unnecessary procedures and prolong morbidity.
Giridhar P, Kalamangalam, Nis, Haq, Simon J, Ellis   +2 more
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Spontaneous intracranial hypotension

British Journal of Hospital Medicine, 2006
A 47-year-old woman presented with a 4-week history of headaches. These were present from waking, and the preceding night's sleep had been disturbed by a sudden popping sensation associated with tinnitus. She described pounding frontal headaches on sitting or standing which were associated with nausea and vomiting, but relieved with bed rest. She had a
E M, Nour, T J, Charles, R P, White
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Spontaneous intracranial hypotension

Current Pain and Headache Reports, 2007
Spontaneous intracranial hypotension (SIH) is caused by leakage of cerebrospinal fluid (CSF), with resultant CSF hypovolemia and intracranial hypotension. Although in some patients SIH may be preceded by minor trauma, it often occurs in the absence of any identifiable initiating event. Orthostatic headache is the primary clinical manifestation, usually
Todd J, Schwedt, David W, Dodick
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Spontaneous Intracranial Hypotension

Obstetric Anesthesia Digest, 2021
(N Engl J Med. 2021;385:2173–2178) Spontaneous intracranial hypotension presents as below-normal cerebrospinal fluid (CSF) due to CSF leakage from an unknown cause. While spontaneous intracranial hypotension is treatable, it is difficult to diagnose.
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Chronic intracranial hypotension

Journal of Clinical Neuroscience, 1998
Acute intracranial hypotension can occur following lumbar puncture or a fall, and sometimes spontaneously. Most cases resolve within weeks or months but some require surgical repair of the defect causing leakage of cerebrospinal fluid (CSF). It is conceivable that such leaks could become chronic if the defect is incompletely sealed.
R A, Mackenzie, A K, Lethlean, R, Shnier, P W, Blum   +3 more
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Spontaneous Intracranial Hypotension

Continuum, 2001
Spontaneous intracranial hypotension results from CSF volume depletion, nearly always from spontaneous CSF leaks. Spontaneous intracranial hypotension is increasingly diagnosed in practice; the number of atypical, unconfirmed, and doubtful cases is also increasing, as are treatment failures. These confront neurologists and create many challenges.
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