Results 171 to 180 of about 968,189 (263)

Olfactory Tuft Cells Are Critical to Basal Inflammation, Innate Immune Response to Viral Infection, and Modulation of Quiescent Stem Cell Activation, Proliferation and Differentiation

open access: yesCell Proliferation, EarlyView.
Olfactory tuft cells communicate with other mucosal cells in maintaining olfactory epithelial immunity and sensory function. Disruption of tuft cell signalling pathways causes a skewed immune response, leading to heightened inflammation and increased activation of the quiescent stem cells, which are further exacerbated by viral infection.
Sai‐Sai Zhang   +7 more
wiley   +1 more source

Exploring the Impact of Ultrasonic Activation on the Antimicrobial Activity of Root Canal Sealers: A Year‐Long Ex Vivo Investigation Into Bacterial Survival

open access: yesInternational Endodontic Journal, EarlyView.
ABSTRACT Aim This study investigated the long‐term survival of bacteria in root canals obturated using two endodontic sealers—AH Plus Jet (AHP) and BioRoot RCS (BCS)—while also assessing the influence of ultrasonic agitation on their antimicrobial effectiveness.
F. L. P. Viana   +7 more
wiley   +1 more source

Genome assembly of Elcysma westwoodi provides insights into host‐plant cyanogenic glucoside detoxification and chemical defence gene evolution

open access: yesInsect Molecular Biology, EarlyView.
A chromosome‐level genome of Elcysma westwoodi was assembled. The genome encodes 272 detoxification‐related genes. A single‐copy bCAS was retained for cyanide detoxification. CYP405A and CYP332A were recruited into cyanogenic defence evolution. UGT33A retained a conserved GT‐B fold and catalytic domains.
Minyoung Choi, Murtaza Khan, Juil Kim
wiley   +1 more source

Hypophosphataemia in adults: approach to diagnosis and management

open access: yesInternal Medicine Journal, EarlyView.
Abstract Hypophosphataemia is a common electrolyte disorder which can have clinically significant consequences for bone, muscle, neurological and haematological function. Its causes range from transient transcellular shifts in hospitalised patients to chronic renal phosphate‐wasting disorders mediated by fibroblast growth factor 23 (FGF23).
Kajanan Parameshwaran   +2 more
wiley   +1 more source

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