Results 131 to 140 of about 1,782 (159)

A Gene for Primary Congenital Glaucoma Is Not Linked to the Locus on Chromosome lq for Autosomal Dominant Juvenile-Onset Open Angle Glaucoma

Journal of Glaucoma, 1996
Kent L. Anderson, Richard A. Lewis, Bassem A. Bejjani, Lisa Baird, Brith Otterud, RKarim F. Tomey, William F. Astle, David Dueker, Mark Leppert, James R. Lupski   +9 more
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Modified viscotrabeculotomy versus modified trabeculotomy in late-onset primary congenital, juvenile, and secondary open-angle glaucoma

European Journal of Ophthalmology
Ghasem Fakhraie, Shabnam Ansari, Seyed Mehdi Tabatabaei, Nikoo Hamzeh, Zakieh Vahedian, Alireza Beikmarzehei   +5 more
openalex   +3 more sources

Dorzolamide, in combination with timolol, improves retinal blood flow in patients with juvenile-onset primary open-angle glaucoma,

Inpharma Weekly, 2007
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Extent of asymmetry and unilaterality among juvenile onset primary open angle glaucoma patients.

Clinical & experimental ophthalmology, 2012
Juvenile onset open angle glaucoma (JOAG) due to its rarity is not well characterized. We aimed to assess the extent of interocular asymmetry of baseline intraocular pressure (IOP), disc morphometry and visual field defects at presentation in patients with primary JOAG.
Viney, Gupta, Shikha, Gupta, Munish, Dhawan, Ajay, Sharma, Kulwant S, Kapoor, Ramanjit, Sihota   +5 more
openaire   +1 more source

A gene for primary congenital glaucoma is not linked to the locus on chromosome 1q for autosomal dominant juvenile-onset open angle glaucoma.

Journal of glaucoma, 1997
Primary congenital glaucoma is an uncommon autosomal recessive condition that results from a developmental defect in the trabecular meshwork and anterior chamber angle, manifesting in the neonatal or infantile period with increased intraocular pressure, corneal enlargement and edema, and optic nerve cupping with consequent loss of vision.
K L, Anderson, R A, Lewis, B A, Bejjani, L, Baird, B, Otterud, K F, Tomey, W F, Astle, D K, Dueker, M, Leppert, J R, Lupski   +9 more
openaire   +1 more source

Novel TIGR/MYOC mutations in families with juvenile onset primary open angle glaucoma

2019
Mutations in the trabecular meshwork induced glucocorticoid response protein (TIGR) or myocilin (MYOC) has recently been shown to cause juvenile onset primary open angle glaucoma (JOAG). In this study, we identified two new mutations (Asp380Ala and Ser502Pro) in two British families and another (Pro370Leu) in a French-Canadian family.
Stoilova D., Child A., Brice G., Desai T., Barsoum-Homsy M., Ozdemir N., Chevrette L.   +6 more
openaire   +1 more source

Juvenile onset primary open-angle glaucoma: three case studies and review.

Journal of the American Optometric Association, 1999
Common clinical characteristics of juvenile onset primary open-angle glaucoma (JPOAG) include increased intraocular pressure, optic nervehead damage, visual-field loss, and a normal-appearing iridocorneal angle by gonioscopy. Histologic analysis of the angle structures may show varying developmental abnormalities that are observed to be less obvious as
openaire   +1 more source

Juvenile-onset open-angle glaucoma – A clinical and genetic update

Survey of Ophthalmology, 2022
Harathy Selvan, Shikha Gupta, Janey L Wiggs   +2 more
exaly  

Myocilin Mutation N480K Leads to Early Onset Juvenile and Adult-onset Primary Open Angle Glaucoma in a Six Generation Family

, 2023
Mohideen Abdul Kader, Bharanidharan Devarajan, Saravanan Vijayan, Rengappa Ramakrishnan, Periasamy Sundaresan, Mohammed Sithiq Uduman, Subbiah R. Krishnadas, Kuppamuthu Dharmalingam   +7 more
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Identification of new TIGR mutations in juvenile-onset primary open angle glaucoma (JOAG) families from different populations.

2019
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Stoilova, D, Child, A, Fleck, BW, Barsoum-Homsy, M, Ozdemir, N, Brice, G, Crick, RP   +6 more
openaire   +1 more source