Results 111 to 120 of about 130,409 (304)

Esophageal cancer presenting with atrial fibrillation: A case report [PDF]

open access: yes, 2008
Introduction Atrial fibrillation was previously reported in patients with esophageal cancer as a complication of total esophagectomy or photodynamic therapy. Here, we propose that atrial fibrillation may also be caused by external compression of the left
Alix Dufresne   +15 more
core   +3 more sources

KLF1 Promotes Cardiomyocyte Proliferation and Heart Regeneration Through Regulation of Wnt/β‐Catenin Signaling Pathway

open access: yesAdvanced Science, EarlyView.
In KLF1‐on (neonatal and Klf1 overexpression hearts) models, KLF1 regulates Wnt/β‐catenin signaling pathway and the expression of cell proliferation‐related genes via transcriptional and epigenetic mechanisms, which induces cardiomyocyte proliferation and cardiac regeneration in mice after myocardial infarction.
Yanglin Hao   +19 more
wiley   +1 more source

Prevalence of congenital heart defects in Europe, 2008–2015: A registry‐based study

open access: yesBirth Defects Research, Volume 114, Issue 20, Page 1404-1416, December 1, 2022., 2022
Abstract Background The total prevalence of congenital heart defects (CHDs) varies by populations and over time. Studies that examine trends in the prevalence of CHD in different regions may shed light on our understanding of the occurrence of CHD and the impact of different risk factors.
Chrysovalanto Mamasoula   +27 more
wiley   +1 more source

Evidence that conflict regarding size of haemodynamic response to interventricular delay optimization of cardiac resynchronization therapy may arise from differences in how atrioventricular delay is kept constant. [PDF]

open access: yes, 2014
Aims: Whether adjusting interventricular (VV) delay changes haemodynamic efficacy of cardiac resynchronization therapy (CRT) is controversial, with conflicting results.
Francis, DP   +9 more
core   +2 more sources

Huge leiomyosarcoma in the left atrium

open access: yesHerz, 2013
Abstract Not ...
Kılıç, İsmail Doğu   +5 more
openaire   +7 more sources

GLS1‐Mediated Redundancy in Glutamate Accelerates Arterial Calcification via Activating NMDAR/Ca2+/β‐Catenin Pathway

open access: yesAdvanced Science, EarlyView.
GLS1 is a novel contributor to arterial calcification. GLS1‐catalyzed glutamate exerts the promoting effects on osteogenic reprogramming in arteries. NMDAR, a glutamate receptor, is also activated and overexpressed during arterial calcification.
Ziting Zhou   +18 more
wiley   +1 more source

Analysis of the course and the ostium of the oblique vein of the left atrium [PDF]

open access: yes, 2000
Considerating the origin of the coronary sinus and the oblique vein of the left atrium, both are remnants of the left horn of the embryonal venous sinus. The studies were carried out on 100 human cadaver hearts.
Cendrowska-Pinkosz, Monika   +1 more
core   +1 more source

Left atrial voltage, circulating biomarkers of fibrosis, and atrial fibrillation ablation. A prospective cohort study. [PDF]

open access: yes, 2018
Aims To test the ability of four circulating biomarkers of fibrosis, and of low left atrial voltage, to predict recurrence of atrial fibrillation after catheter ablation.
A Verma   +28 more
core   +6 more sources

Massive calcification of giant left atrium in a rheumatic mitral stenosis patient: A case report

open access: yesRespiratory Medicine Case Reports, 2019
Giant left atrium is a condition characterized by an extreme enlargement of the left atrium with a diameter more than 80 mm and it is usually associated with long standing rheumatic mitral valve disease.
Yong Luo   +3 more
doaj  

Post prandial and nocturnal recurrent acute heart failure caused by a large hiatal hernia

open access: yesJournal of Acute Disease, 2012
We report a case of left heart failure caused by a sliding hiatus hernia compressing on left atrium. A 95-year-old woman was admitted with recurrent episodes of shortness of breath and chest pain.
Abdurrahim Dusak
doaj   +1 more source

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