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Clinical features of limbic encephalitis with LGI1 antibody
, 2017 Meiling Wang +5 more
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Remote digital cognitive assessment reveals cognitive deficits related to hippocampal atrophy in autoimmune limbic encephalitis: a cross-sectional validation study. [PDF]
EClinicalMedicineShibata K +8 more
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Striatum Involvement in LGI1 Limbic Encephalitis
Clinical Psychopharmacology and Neuroscience, 2018 openaire +5 more sources
The diagnosis and treatment of limbic encephalitis
Acta Neurologica Scandinavica, 2012 The term limbic encephalitis (LE) was first introduced in 1968. While this disease was initially considered rare and is often fatal with very few treatment options, several reports published in the last decade provide a better description of this condition as well as possible causes and some cases of successful treatment.
Fredrik Asztely, Eva Kumlien
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Limbic encephalitis and psychosis
General Hospital Psychiatry, 2013To report a case of limbic encephalitis (LE) presenting with psychosis.Case report.A woman with LE initially presented with acute psychotic symptoms. Magnetic resonance imaging (MRI) showed hyperintensity in bilateral temporal lobes. Paraneoplastic and autoimmune antibodies were negative, but symptoms resolved after starting methylprednisolone.A ...
Paula Tabares +5 more
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2022
Limbic encephalitis (LE) is a clinical syndrome defined by subacutely evolving limbic signs and symptoms with structural and functional evidence of mediotemporal damage in the absence of a better explanation than an autoimmune (or paraneoplastic) cause. There are features common to all forms of LE.
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Limbic encephalitis (LE) is a clinical syndrome defined by subacutely evolving limbic signs and symptoms with structural and functional evidence of mediotemporal damage in the absence of a better explanation than an autoimmune (or paraneoplastic) cause. There are features common to all forms of LE.
openaire +2 more sources
Paraneoplastic Limbic Encephalitis
Psychosomatics, 2009Paraneoplastic limbic encephalitis (PLE) is a syndrome characterized by the development of neuropsychiatric symptoms due to a misdirected tumor-activated antibody response that damages host neural tissue. Definitive treatment relies on the identification and ablation of the underlying tumor.
Alison R. Foster +3 more
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