Results 191 to 200 of about 31,019,635 (353)
Optimal Sequential Fusion Kalman Filter for Multi-Sensor Linear Systems with Noise Cross-Correlated. [PDF]
Huang W, Wen C.
europepmc +1 more source
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira +14 more
wiley +1 more source
Stability Analysis of Discrete-time Switched Linear Systems with Parametric Uncertainties
Mohamad Hosein Shafiei +1 more
openalex +2 more sources
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian +10 more
wiley +1 more source
Effort-Optimized, Accuracy-Driven Labelling and Validation of Test Inputs for DL Systems: A Mixed-Integer Linear Programming Approach [PDF]
Mohammad Hossein Amini +2 more
openalex +1 more source
Solving linear systems on quantum hardware with hybrid HHL+. [PDF]
Yalovetzky R +3 more
europepmc +1 more source
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath +13 more
wiley +1 more source
Data-Driven Superstabilization of Linear Systems under Quantization. [PDF]
Miller J +3 more
europepmc +1 more source
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source

