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LPAR1 Promotes Activation of Pathways Alveolar Hypercoagulation and Fibrinolytic Inhibition in ARDS via NF-κB Signaling Pathway. [PDF]

FASEB Bioadv
Liu Y, Chen X, Liu M, Yuan J, Li Q, Li L, Dong Q, Tao J, Shen F, Xiao C.   +9 more
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LIPOPOLYSACCHARIDE RECOGNITION, CD14, AND LIPOPOLYSACCHARIDE RECEPTORS

Infectious Disease Clinics of North America, 1999
The ability of a host to sense invasion by a pathogenic organism, and to respond appropriately to control infection, is paramount to survival. To that end, an array of receptors and binding proteins has evolved as part of the innate immune system to detect Gram-negative bacteria.
R R, Ingalls, H, Heine, E, Lien, A, Yoshimura, D, Golenbock   +4 more
openaire   +2 more sources

Lipopolysaccharide-Dependent and Lipopolysaccharide-Independent Pathways of Monocyte Desensitisation to Lipopolysaccharides

Journal of Leukocyte Biology, 1991
Abstract The present study demonstrates that with time in culture blood monocytes (MO) lose their ability to express procoagulant activity (PCA) and secrete tumor necrosis factor-alpha (TNFα) in culture medium in response to lipopolysaccharide (LPS) stimulation.
A Y, Annenkov, F S, Baranova
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Supported Lipopolysaccharide Bilayers

Langmuir, 2012
In this report, the formation of supported lipopolysaccharide bilayers (LPS-SLBs) is studied with extracted native and glycoengineered LPS from Escherichia coli ( E. coli ) and Salmonella enterica sv typhimurium ( S. typhimurium ) to assemble a platform that allows measurement of LPS membrane structure and the detection of membrane tethered ...
Stefan, Kaufmann, Karin, Ilg, Alireza, Mashaghi, Marcus, Textor, Bernard, Priem, Markus, Aebi, Erik, Reimhult   +6 more
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Lipopolysaccharides ofShigella sonnei

Applied Biochemistry and Microbiology, 2000
Immunobiological properties of native lipopolysaccharides (LPS) from virulent and avirulent strains of Shigella sonnei bacteria (LPS-V and LPS-A, respectively) were studied. In avirulent bacteria, LPS-V induced immunosuppressive activity specific of the virulent strain. LPS of the avirulent strain, whereas LPS-A lacked this property.
E V, Borisova, V A, Borisov
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PATHOBIOLOGY OF LIPOPOLYSACCHARIDE

Journal of Toxicology and Environmental Health, 1997
Lipopolysaccharide is a component of the gram-negative bacterial cell wall that is responsible for 25,000-50,000 deaths in the United States each year. The sequelae of gram-negative infection and septicemia leading to death include fever, hypotension with inadequate tissue perfusion, and disseminated intravascular coagulation.
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