Results 161 to 170 of about 34,480 (242)

Case report: CMR work-up for a case of long QT syndrome, dilated left ventricle, and hypertension-when incidental findings unmasked the fundamental issue. [PDF]

Eur Heart J Case Rep
Aye N, Velegraki E, Mohiaddin RH.
europepmc   +1 more source

14‐3‐3 proteins: Regulators of cardiac excitation–contraction coupling and stress responses

The Journal of Physiology, Volume 604, Issue 16, Page 6619-6638, 15 August 2026.
Abstract figure legend 14‐3‐3 protein interactions in cardiac regulation. Schematic representation of 14‐3‐3 binding partners in excitation–contraction coupling, transcriptional regulation/development and stress response pathways. Asterisks indicate targets where the exact 14‐3‐3 binding site is unknown.
Heather C. Spooner, Rose E. Dixon
wiley   +1 more source

Congenital Long QT Syndrome Unmasked by COVID-19 Infection Presenting as Ventricular Tachycardia Storm. [PDF]

JACC Case Rep
Aoki H, Enomoto Y, Wada M, Kubota S, Kato N, Hara H, Hiroi Y.   +6 more
europepmc   +1 more source

Repolarization adaptation to rapid change in heart rate in human models – a review

The Journal of Physiology, Volume 604, Issue 16, Page 6675-6690, 15 August 2026.
Abstract figure legend This review focuses on non‐invasive assessment of repolarization duration and dispersion (heterogeneity) adaptation to change in heart rate (HR). HR was increased incrementally by left atrial pacing during an electrophysiology (EP) study and by a bolus injection of atropine and in a step up/down fashion by repeated right atrial ...
Lennart Bergfeldt, Karl‐Jonas Axelsson, Pia Dahlberg, Farzad Vahedi, Gunilla Lundahl, Lennart Gransberg   +5 more
wiley   +1 more source

Sudden death across brain and heart: cardio-cerebral ion channel dysfunction as a potential risk-modifying mechanism linking epilepsy and long QT syndrome. [PDF]

Front Neurol
Zhang J, Chen Q, Ma Z, Luo K, Ben X.
europepmc   +1 more source

Small‐conductance Ca2⁺‐activated K⁺ channels in cardiac excitation–contraction coupling: Bridging mitochondria, sarcolemma and antiarrhythmic therapy

The Journal of Physiology, Volume 604, Issue 16, Page 6691-6703, 15 August 2026.
Abstract figure legend Mitochondrial SK channel enhancement reduces cardiac arrhythmia trigger. Spontaneous sarcoplasmic reticulum (SR) Ca2+ release via hyperactive RyR2s underlies an increased arrhythmia trigger, promoting early and delayed afterdepolarizations during stress. Hyperactive RyR2s causes rise in cytosolic [Ca2+] during diastole. Clearance
Dmitry Terentyev, Alexey V. Glukhov, Vladimir Bogdanov, Aleksei S. Averin, Roland Veress, Bum‐Rak Choi, Shanna Hamilton, Andriy E. Belevych   +7 more
wiley   +1 more source

Spatial and temporal dispersion of ventricular repolarization in pediatric patients with congenital long QT syndrome. [PDF]

HeartRhythm Case Rep
Dai L, Zhang C, Ramdat Misier NL, van Schie MS, Bear L, Haissaguerre M, Taverne YJHJ, de Groot NMS.   +7 more
europepmc   +1 more source

Population-Based Study Found Low Risk of Misdiagnosing Long QT Syndrome as Breath-Holding Spells in Swedish Children. [PDF]

Acta Paediatr
Schmidt SH, Jeremiasen I, Eklund EA, Pronk CJ.   +3 more
europepmc   +1 more source

Long QT Syndrome With KCNH2/SCN5A Dual Gene Mutation Leading to Electrical Storm. [PDF]

JACC Case Rep
Ding Q, Lu L, Li S, Guo M.
europepmc   +1 more source