Results 181 to 190 of about 104,993 (291)
Correction to: Multicentre study of the role of lumbar puncture in the diagnosis of spontaneous subarachnoid haemorrhage. [PDF]
Thompson D +7 more
europepmc +1 more source
This study identifies CRISPLD2 as a key protector against IVDD. By regulating ferroptosis through the CRISPLD2–miR‐548I–IL17A axis, CRISPLD2 maintains NPCs homeostasis and reduces oxidative stress. Restoring CRISPLD2 expression effectively alleviates disc degeneration and highlights a promising therapeutic strategy for discogenic low back pain ...
Yangyang Shi +11 more
wiley +1 more source
Video-Assisted Instruction Versus Simulation-Based Mastery Learning for Lumbar Puncture Skill Development in Undergraduates: A Randomized Study. [PDF]
Bhat A +3 more
europepmc +1 more source
A mechanically robust, ROS‐responsive hydrogel is engineered to treat intervertebral disc degeneration by targeting the NF‐κB–LCN2 inflammatory feedback loop. By delivering wedelolactone specifically within oxidative niches, this system disrupts macrophage–disc cell crosstalk.
Zimei Wu +10 more
wiley +1 more source
Reactive Pleocytosis After Repeated Lumbar Puncture-Implications for Clinical Practice. [PDF]
Schmidauer M +13 more
europepmc +1 more source
Tracking early cognitive decline in preclinical AD with brain MRI similarity
Abstract INTRODUCTION Early detection of neuroanatomical changes in preclinical Alzheimer's disease (AD) is critical for timely intervention. However, conventional magnetic resonance imaging (MRI) and fluid biomarkers often lack sensitivity to subtle structural alterations in early disease stages. METHODS To identify early brain alterations, we applied
Jiawei Sun +6 more
wiley +1 more source
Cerebral Venous Thrombosis Following a Lumbar Puncture in a Patient Using NuvaRing. [PDF]
Altaf R +5 more
europepmc +1 more source
Measurements of Biogenic Amines and Metabolites in the CSf of Suicide Victims and Nonsuicides [PDF]
CMB Pare +7 more
core +1 more source
Alpha‐synuclein co‐pathology in a real‐world early Alzheimer's disease cohort
Abstract BACKGROUND Most Alzheimer's disease (AD) cases show mixed pathology, with α‐synuclein (αSyn) aggregates present in a substantial proportion. The cerebrospinal fluid (CSF) α‐synuclein seed amplification assay (αS‐SAA) enables in vivo detection of pathogenic αSyn aggregates, but its clinical significance remains unclear. METHODS We prospectively
Tamara Shiner +15 more
wiley +1 more source

