Results 281 to 290 of about 53,357 (343)
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The Luteal Phase Defect

Fertility and Sterility, 1976
In summary, the luteal phase defect is a deficiency of corpus luteum progesterone steroidogenesis, either in amount or duration, or both. The clinical manifestations include either primary infertility or repeated first trimester abortions. The diagnosis can only be made clinically on the basis of a well-timed endometrial biopsy that is read ...
Edward E. Wallach   +1 more
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Luteal-phase endocrinology

Reproductive BioMedicine Online, 2009
The corpus luteum is formed from the pre-ovulatory follicle under the action of the mid-cycle LH surge. LH is the main luteotrophic hormone in women controlling luteal structure and function during the normal menstrual cycle. Local factors, however, including progesterone are also involved.
Ioannis E. Messinis   +2 more
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Luteal-Phase Defect

Clinical Obstetrics and Gynecology, 1991
Corpus luteum function depends on normal granulosa and theca cell components, which in turn are stimulated by an adequate luteinizing hormone (LH) surge in both duration and amplitude of pulses with LH residual pulses of adequate amplitude during the 14-day luteal span.
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Luteal Phase Dysfunction

Clinics in Obstetrics and Gynaecology, 1981
SUMMARY AND CONCLUSIONS Direct and indirect measures of progesterone secretion are the basis for assessing corpus luteum function. Despite methodological advances such as radioligand binding assays for the hormone, the frequency with which corpus luteum defects result in infertility or early abortion remains a matter of contention.
Gere S. diZerega, Griff T. Ross
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Endocrinology in luteal phase and implantation

British Medical Bulletin, 1990
The normality of the luteal phase after superovulation depends on the method and adequacy of the stimulation regimen. In comparison with natural cycles, the luteinisation of multiple follicles produces higher concentrations of steroids in blood, and soon after ovulation a correct ratio of serum oestradiol (E2) to progesterone is critical to establish ...
Smitz, Johan   +2 more
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The short luteal phase and infertility

BJOG: An International Journal of Obstetrics & Gynaecology, 1984
Summary. The distribution of the length of the luteal phase was investigated in 187 women with regular, apparently ovulatory menstrual cycles of whom 95 had unexplained infertility and the other 92 comprised a group of healthy volunteer subjects. If the short luteal phase is associated with infertility it might be expected t o occur more frequently in
Stephen K. Smith   +3 more
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The defective luteal phase

Human Reproduction, 1988
A defective luteal phase (DLP) results from a relative deficiency in secretion of progesterone by the corpus luteum. Approximately 30% of normal women show on biopsy an occasional DLP, while only 7-14% of infertile patients have a recurrent DLP. Endometrial biopsy remains the classical way to diagnose an inadequate luteal phase.
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Endometrial receptivity and the luteal phase

Current Opinion in Obstetrics and Gynecology, 1992
Endometrial receptivity to the implanting blastocyst determines whether pregnancy occurs. Whether fertilization is initiated in vivo or in vitro, the receptivity of the luteal-phase endometrium results from the input of interacting hormonal, growth, and immunologic factors.
Anne Colston Wentz, Albert J. Peters
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Individualized luteal phase support

Current Opinion in Obstetrics & Gynecology, 2019
Purpose of review The aim of this review is to summarize the different aspects of luteal phase deficiency in IVF treatment and the possibilities of individualized luteal phase support. Recent findings After the application of human chorionic gonadotrophin (hCG) for final oocyte ...
Barbara Lawrenz   +2 more
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Endometriosis and Luteal Phase Defect

Asia-Oceania Journal of Obstetrics and Gynaecology, 1993
AbstractIt has been reported that there exists a correlation between endometriosis and the luteal phase defect (LPD) as one of the determinants in infertility. In some theories, the hampered maturation of the corpus luteum is believed to be caused by the reduction of LH receptors, increase in prolactin, prostaglandin and macrophages in endometriosis ...
F. A. Moeloek, Endy Moegny
openaire   +3 more sources

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