Results 181 to 190 of about 1,251,059 (351)

European Society for Medical Oncology (ESMO) Gastrointestinal Cancers congress 2025. [PDF]

Lancet Reg Health Eur
Nedic I.
europepmc   +1 more source

European society for medical oncology

, 2000

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β‐TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1

Molecular Oncology, EarlyView.
Low levels of β‐TrCP (Panel A) allow the accumulation of BRCA1 and CtIP, which facilitate the repair of cisplatin‐induced DNA damage via homologous recombination (HR) and promote tumor cell survival. In contrast, high β‐TrCP expression (Panel B) leads to BRCA1 and CtIP degradation, impairing HR repair, resulting in persistent DNA damage and apoptosis ...
Rocío Jiménez‐Guerrero, Alejandro Belmonte‐Fernández, Mónica González‐Moreno, Laura Rodríguez‐Cordero, Begoña Pérez‐Valderrama, Joaquín Herrero‐Ruíz, Francisco Romero, Carmen Sáez, Miguel Á. Japón   +8 more
wiley   +1 more source

Implementing Ryan's Law on an Inpatient Medical Oncology Unit. [PDF]

Clin J Oncol Nurs
McKaig A, Varilla Ridad AR, Bell A, McParlane R, Quirch M.   +4 more
europepmc   +1 more source

Über 5-FU hinaus: Zukünftige therapeutische Ansätze zur Behandlung des kolorektalen Karzinoms. Zum Vortrag «Beyond 5-FU: Future Directions in the Treatment of Colorectal Cancer»von D.G. Haller (University of Pennsylvania Cancer Center Medical und Hematology-Oncology Outpatient Practice, Hospital of the University of Pennsylvania, Philadelphia, PA

, 2000
Daniel G. Haller
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On the Deluge of Unsubscribed Medical Oncology Information [PDF]

, 2005
David M. Mintzer, Lauren Chmielewski
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The critical role of DNA damage‐inducible transcript 4 (DDIT4) in stemness character of leukemia cells and leukemia initiation

Molecular Oncology, EarlyView.
Stemness properties, including quiescence, self‐renewal, and chemoresistance, are closely associated with leukemia relapse. Here, we demonstrate that DNA damage‐inducible transcript 4 (DDIT4) is induced in the hypoxic bone marrow niche and is essential for maintaining the stemness of AML1‐ETO9a leukemia cells.
Yishuang Li, Zhijie Cao, Haiyan Xing, Zhenya Xue, Wenbing Liu, Jiayuan Chen, Yihan Mei, Runxia Gu, Hui Wei, Shaowei Qiu, Min Wang, Qing Rao, Jianxiang Wang   +12 more
wiley   +1 more source

The European Society for Medical Oncology (ESMO) [PDF]

, 2005

openalex   +1 more source

Unveiling unique protein and phosphorylation signatures in lung adenocarcinomas with and without ALK, EGFR, and KRAS genetic alterations

Molecular Oncology, EarlyView.
Proteomic and phosphoproteomic analyses were performed on lung adenocarcinoma (LUAD) tumors with EGFR, KRAS, or EML4–ALK alterations and wild‐type cases. Distinct protein expression and phosphorylation patterns were identified, especially in EGFR‐mutated tumors. Key altered pathways included vesicle transport and RNA splicing.
Fanni Bugyi, Mirjam Balbisi, Simon Sugár, Lóránd Váncza, Eszter Regős, Ilona Kovalszky, Ibolya Laczó, Tünde Harkó, Gábor Kecskeméti, Zoltán Szabó, Judit Moldvay, László Drahos, Lilla Turiák   +12 more
wiley   +1 more source

Sexual health in cancer care: a narrative review and position statement from the Italian Association of Medical Oncology (AIOM). [PDF]

ESMO Open
Rossi A, Leone AG, Lambertini M, Sperti E, Cassani C, Vetromile A, Barni S, De Giorgi A, Preti EP, Secondino S, Silvestris N, Berardi R, Brunello A, Curigliano G, La Verde N, Latiano TP, Pietrantonio F, Tiseo M, Di Maio M, Perrone F, Cinieri S, Fabi A, Associazione Italiana Oncologia Medica (AIOM).   +22 more
europepmc   +1 more source