Results 181 to 190 of about 26,131 (235)

Impact of incisura biopsy on the surveillance of precursor lesions and gastric cancer risk assessment. [PDF]

open access: yesGastroenterol Rep (Oxf)
Cantú-Germano E   +9 more
europepmc   +1 more source

The OLGIMA system for gastric cancer risk assessment. A useful method based on the histological Sydney consensus. [PDF]

open access: yesHistopathology
Delgado Guillena PG   +29 more
europepmc   +1 more source

Hepatocyte Antigen as a Marker of Intestinal Metaplasia

open access: yesThe American Journal of Surgical Pathology, 2003
Intestinal metaplasia is a histologic hallmark of Barrett's esophagus and chronic gastritis. Intestinal metaplasia may progress to dysplasia or carcinomas without proper treatment. Most cases of intestinal metaplasia are easily recognized on hematoxylin and eosin-stained sections. However, some cases of intestinal metaplasia may be hard to recognize if
Peiguo G, Chu   +2 more
openaire   +3 more sources

Operative Link for Gastritis Assessment gastritis staging incorporates intestinal metaplasia subtyping

open access: yesHuman Pathology, 2011
Both sulfomucin-type intestinal metaplasia (ie, types II and III intestinal metaplasia, colonic-type intestinal metaplasia) and gastritis in Operative Link for Gastritis Assessment stages III and IV are associated with an increased risk of intestinal ...
Massimo Rugge   +2 more
exaly   +2 more sources

Gastric intestinal metaplasia revisited: function and regulation of CDX2

open access: yesTrends in Molecular Medicine, 2012
Intestinal metaplasia of the stomach is a preneoplastic lesion that appears following Helicobacter pylori infection and confers increased risk for gastric cancer development.
Rita Barros   +2 more
exaly   +2 more sources

Oesophageal intestinal metaplasia

Digestive and Liver Disease, 2000
An alarmingly rapid rise in adenocarcinoma at the level of the gastro-oesophageal junction and distal oesophagus has been noted over the past two decades l 2. Intestinal metaplasia (IM) is considered to be the main precursor lesion for such adenocarcinomas.
Tytgat, G. N., van Sandick, J. W.
openaire   +3 more sources

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