Results 251 to 260 of about 354,115 (299)

CHCHD10 Mitigates Alzheimer's Disease‐Related Phenotypes in Association With Epigenetic Remodeling in Directly Reprogrammed Neurons

Advanced Science, EarlyView.
CHCHD10 loss in Alzheimer's disease is associated with mitochondrial dysfunction, epigenomic disruption, and tau pathology. Restoration of CHCHD10 shifts DNA methylation toward a non‐disease state and reduces tau and amyloid pathology, with KATNAL2 acting as a downstream effector.
Teresa M. Thomas, Ching‐Yao Yang, Kexin Zhang, Liam Wetzel, Dina Bugybayeva, Julia Ferguson, Md Mahmudul Hasan, William Samsa, Nandita Patil, Anshul Dash, Tarini Gowda, Xiongwei Zhu, Feixiong Cheng, Tian Liu   +13 more
wiley   +1 more source

Piecemeal mitochondrial degradation in plants: a coordination between mitochondrial dynamics and mitophagosome formation. [PDF]

Autophagy
Ma J, Li C, Zhuang X.
europepmc   +1 more source

Nanoparticles that target nonamyloid and nontau pathways in Alzheimer's disease. [PDF]

Discov Nano
Alavian F, Hajimohammadi A.
europepmc   +1 more source

Unique mitochondrial carrier has a role in mitochondrial dynamics during Plasmodium falciparum host switching

Boshoven C, Tassan-Lugrezin S, King AC, Kunji ER, Kooij TW.   +4 more
europepmc   +1 more source

Dimethyl fumarate and mitochondrial physiology: implications for neurological disorders. [PDF]

Front Pharmacol
de Oliveira MR.
europepmc   +1 more source

PRRSV subverts host programmed cell death network for immune evasion. [PDF]

Virulence
Ma S, Mao Q, Weng S, Lian K, Jiang Z, Hao J, Zhang K.   +6 more
europepmc   +1 more source

Apolipoproteins L involvement in immunity. [PDF]

J Hum Immun
Pays E.
europepmc   +1 more source

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Biology and Pathology of the Mitochondrion

Ultrastructural Pathology, 1998
The mitochondrion is a highly efficient organelle that is essential for the function of cells. It is currently accepted that mitochondria originated from primitive nonphotosynthetic bacteria that were engulfed by early eucharyotic cells. Most of the normal and pathologic interactions between mitochondria and the cells that contain them can be viewed as
Lloreta J, Sergi Serrano
exaly   +3 more sources