Results 151 to 160 of about 250,975 (359)
Abstract Background Cardiac remodelling, a crucial aspect of heart failure, is commonly investigated in preclinical models by quantifying cardiomyocyte cross‐sectional area (CSA) and microvascular density (MVD) via histological methods, such as immunohistochemistry.
Tamás G. Gergely +14 more
wiley +1 more source
Background The purpose of our prospective study is to investigate the histopathology of Muller’s muscle extracted after Muller’s Muscle-Conjunctival Resection (MMCR) and to find the relationship between histopathological findings and the outcomes of ...
Hassan Asadigandomani +5 more
doaj +1 more source
Hypertrophy of the Masseter Muscles [PDF]
openaire +3 more sources
Aldose Reductase Inhibitor Prevents Hyperproliferation and Hypertrophy of Cultured Rat Vascular Smooth Muscle Cells Induced by High Glucose [PDF]
Kenichi Yasunari +5 more
openalex +1 more source
Rabbit models of heart disease. [PDF]
Human heart disease is a major cause of death and disability. A variety of animal models of cardiac disease have been developed to better understand the etiology, cellular and molecular mechanisms of cardiac dysfunction and novel therapeutic strategies ...
Bers, Donald M, Pogwizd, Steven M
core
This systematic review identified 24 clinical trials of medical device and surgical interventions for HFpEF, with 16 demonstrating at least safety and feasibility, but little evidence of long‐term benefit. Phenotype‐based responses suggest that certain interventions may benefit specific HFpEF subgroups, yet most trials suffered from heterogeneous ...
Kurdo Araz +5 more
wiley +1 more source
Three cases of hypertrophy of the masseteric muscle.
Asako Matsumoto +9 more
openalex +2 more sources
Alternative splicing and cycling kinetics of myosin change during hypertrophy of human smooth muscle cells [PDF]
Ingo Morano +7 more
openalex +1 more source
The emerging role of Nrf2 in heart failure: From cardioprotection to therapeutic approaches
Nrf2 signalling is impaired in heart failure, contributing to oxidative stress, mitochondrial dysfunction, and ferroptosis, which drive neurohormonal activation, inflammation, and ischaemia‐reperfusion injury. This loss of Nrf2 activity exacerbates hypertension, diabetic cardiomyopathy, and cardiotoxicity, accelerating progression to overt heart ...
Emiliano Fiori +9 more
wiley +1 more source

