Results 261 to 270 of about 1,899,851 (400)

Fetal Ductal Constriction, Pulmonary Hypertension and Atrial Septum Mobility

open access: yesJournal of Ultrasound in Medicine, EarlyView.
Objectives Fetuses with ductal constriction (FDC), pulmonary hypertension and right ventricular overload after maternal exposure to PGE2 inhibitors—nonsteroidal anti‐inflammatory drugs (NSAIDs) or polyphenols (PF)—have increased septum primum excursion index (SPEI), the ratio between maximal septum primum displacement and left atrial diameter.
Paulo Zielinsky   +5 more
wiley   +1 more source

Masticatory muscle hypertrophy

open access: yes, 2019
Mostafa Elfeky   +2 more
openaire   +1 more source

Adjunctive Botulinum Toxin Type A for Bruxism and Sweat‐Worsened Facial Blistering in Recessive Dystrophic Epidermolysis Bullosa: A Case Series

open access: yesJEADV Clinical Practice, EarlyView.
Capsule Summary Bruxism and sweat‐induced facial blisters can affect patients with Recessive Dystrophic Epidermolysis Bullosa (RDEB) quality of life (QoL). We present the outcomes of orofacial BTX‐A infiltrations to treat these conditions. Three patients with severe RDEB, bruxism and microstomia received BTX‐A infiltrations (50U‐100U) in masseters and ...
Susanne Krämer   +4 more
wiley   +1 more source

Muscle RING finger-1 attenuates IGF-I-dependent cardiomyocyte hypertrophy by inhibiting JNK signaling

open access: green, 2014
Kristine M. Wadosky   +5 more
openalex   +1 more source

MiR‐145‐5p Attenuates Doxorubicin‐Induced Heart Injury Through Targeting Cardiomyocyte Pyroptosis

open access: yesThe Kaohsiung Journal of Medical Sciences, EarlyView.
ABSTRACT Doxorubicin (DOX), a potent anthracycline chemotherapeutic, exhibits dose‐dependent cardiotoxicity that limits its clinical utility. Although miR‐145‐5p demonstrates cardioprotective properties in cardiovascular diseases, its role in DOX‐induced cardiomyopathy remains undefined.
Xing‐Tao Chen   +4 more
wiley   +1 more source

TET3‐Mediated m5C Modification of CCAT2 Accelerates Cardiac Microvascular Endothelial Cell Damage in Acute Coronary Syndrome

open access: yesThe Kaohsiung Journal of Medical Sciences, EarlyView.
ABSTRACT Acute coronary syndrome (ACS) is a clinical syndrome involving myocardial ischemia. This study aimed to elucidate the mechanism of TET3 in ACS‐induced CMEC damage, thereby identifying a new target for ACS treatment. The expression of TET3 in ACS patients and healthy subjects was analyzed.
Jun‐Cheng Liu   +5 more
wiley   +1 more source

<i>Rosa rugosa</i> promotes muscle hypertrophy and prevents atrophy in C2C12 myoblasts. [PDF]

open access: yesFood Sci Biotechnol
Kim H   +8 more
europepmc   +1 more source

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