Results 221 to 230 of about 91,972 (285)
MYD88L256P mutation was searched in 79 CIDP, 35 MMN, and 57 controls. It was more frequent in MMN (34%) and CIDP (19%) than in controls (2%). It suggests new pathophysiological hypotheses and new therapeutic approaches for chronic inflammatory neuropathies.
Alexandre Guérémy+11 more
wiley +1 more source
Antimuscle Factor and Antinuclear Factor in Patients with Myasthenia Gravis [PDF]
D. E. McFarlin+2 more
openalex +1 more source
Botulinum Neurotoxins: History, Mechanism, and Applications. A Narrative Review
Botulinum neurotoxins (BoNTs), produced by Clostridium botulinum, bind irreversibly to presynaptic cholinergic nerve terminals and exploit a zinc‐dependent mechanism to cleave SNARE proteins, blocking acetylcholine release and causing flaccid paralysis.
Arik Monash+3 more
wiley +1 more source
Myasthenia Gravis, Hashimoto's Thyroiditis, and Pernicious Anaemia [PDF]
Wolfgang Singer, B. M. Sahay
openalex +1 more source
Case of Myasthenia Gravis, with Affection of the Larynx and Soft Palate
E. D. D. Davis
openalex +1 more source
Sensitivity to Curare by a Patient with Undiagnosed Myasthenia Gravis Syndrome [PDF]
Blossom A. Sanger, Gilbert E. Kinyon
openalex +1 more source