Results 331 to 340 of about 749,314 (382)

A network-based approach to overcome BCR::ABL1-independent resistance in chronic myeloid leukemia. [PDF]

open access: yesCell Commun Signal
Bica V   +15 more
europepmc   +1 more source

Decoding Acute Myeloid Leukemia: A Clinician's Guide to Functional Profiling. [PDF]

open access: yesDiagnostics (Basel)
Iyer P   +4 more
europepmc   +1 more source

Lactate dehydrogenase A-coupled NAD+ regeneration is critical for acute myeloid leukemia cell survival

open access: yes
Erdem A   +8 more
europepmc   +1 more source

Acute Myeloid Leukemia

Hematology/Oncology Clinics of North America, 2008
Acute myeloid leukemia (AML) is a heterogeneous group of leukemias that result from clonal transformation of hematopoietic precursors through the acquisition of chromosomal rearrangements and multiple gene mutations. As a result of highly collaborative clinical research by pediatric cooperative cancer groups worldwide, disease-free survival has ...
Jeffrey E, Rubnitz   +2 more
  +7 more sources

Chronic myeloid leukemia: 2022 update on diagnosis, therapy, and monitoring

American journal of hematology/oncology, 2022
Chronic Myeloid Leukemia (CML) is a myeloproliferative neoplasm with an incidence of 1–2 cases per 100 000 adults. It accounts for approximately 15% of newly diagnosed cases of leukemia in adults.
E. Jabbour, H. Kantarjian
semanticscholar   +1 more source

Acute Myeloid Leukemia

Hematology/Oncology Clinics of North America, 2009
The evolution of acute myeloid leukemia (AML) classification reflects greater understanding of the AML pathogenesis. The 2008 World Health Organization classification incorporated cytogenetic and molecular genetic findings and introduced important prognostic correlations.
Amy, Heerema-McKenney, Daniel A, Arber
openaire   +2 more sources

Chronic myeloid leukemia

Current Opinion in Hematology, 1997
Progress has been made in understanding BCR-ABL-positive leukemias. A new transcript (p230BCR-ABL) has been characterized that is associated with Ph-positive chronic neutrophilic leukemia. The ATM protein appears to be a regulator of ABL activity in response to irradiation damage.
John M. Goldman, Michael W. Deininger
openaire   +4 more sources

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