Inklusionsorienteret NAV praksis?
Caswell, Dorte, Innjord, Aud Kirsten
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NAV-005, a high-affinity MUC16/CA125 antagonist for the treatment of humoral immunosuppressed cancers. [PDF]
Nicolaides NC, Kline JB, Grasso L.
europepmc +1 more source
Biochemical assessment of α-α-subunit interactions of Na<sub>v</sub>1.5 in a heterologous expression system. [PDF]
Iamshanova O +7 more
europepmc +1 more source
mTORC2-Na<sub>v</sub>1.2 signaling drives early hyperexcitability in Alzheimer's disease mouse model. [PDF]
Dvorak NM, Noebels JL.
europepmc +1 more source
Experimental Platform for Screening and Validation of BacNa<sub>v</sub> Gene Therapy Candidates. [PDF]
Wu T, Nguyen HX, Siu YY, Li Y, Bursac N.
europepmc +1 more source
Matrix metalloproteinase activation and TNF upregulation characterize the sclerotic phase of aortic valve disease. [PDF]
Chan-Delgado E +10 more
europepmc +1 more source
Fluorine-free acetonitrile-based electrolytes with anion-reinforced solvation chemistry for sustainable sodium-ion full batteries. [PDF]
Xu X +13 more
europepmc +1 more source
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Nav Channels in Damaged Membranes
2016Sick excitable cells (ie, Nav channel-expressing cells injured by trauma, ischemia, inflammatory, and other conditions) typically exhibit "acquired sodium channelopathies" which, we argue, reflect bleb-damaged membranes rendering their Nav channels "leaky." The situation is excitotoxic because untreated Nav leak exacerbates bleb damage.
C E, Morris, B, Joos
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