Results 191 to 200 of about 60,277 (228)
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Nephrotic Syndrome

Pediatrics In Review, 2022
Nephrotic syndrome (NS) encompasses a variety of disease processes leading to heavy proteinuria and edema. Minimal change disease (MCD) remains the most common primary cause of NS, as well as the most responsive to pharmacologic treatment with often minimal to no chronic kidney disease.
Estefania, Rodriguez-Ballestas   +1 more
openaire   +2 more sources

Nephrotic Syndrome

Primary Care: Clinics in Office Practice, 2020
Nephrotic syndrome is one cause of end-stage kidney disease. Because edema is a common presenting feature and hypertension and dyslipidemia are often present in nephrotic syndrome, it is important for the primary care physician to suspect this entity.
Seth Anthony, Politano   +2 more
openaire   +2 more sources

Childhood nephrotic syndrome

The Lancet, 2023
Idiopathic nephrotic syndrome is the most common glomerular disease in children. Corticosteroids are the cornerstone of its treatment, and steroid response is the main prognostic factor. Most children respond to a cycle of oral steroids, and are defined as having steroid-sensitive nephrotic syndrome.
Marina, Vivarelli   +3 more
openaire   +2 more sources

The nephrotic syndrome

American Heart Journal, 1957
Abstract The nephrotic syndrome is apparently due to continued excretion (and destruction ?) of serum albumin by the kidney. The chief clinical consequences are proteinuria, hypoalbuminemia, edema, lipemia, and susceptibility to infection and, all too often, to terminal renal failure. The nephrotic syndrome is associated with many renal diseases. The
H L, BARNETT, H A, EDER
  +10 more sources

Nephrotic Syndrome

The Indian Journal of Pediatrics, 2012
The pathogenetic basis of idiopathic nephrotic syndrome, a common childhood glomerulopathy, is being explored. While initial evidence supported an imbalance of T helper responses, recent studies suggest alterations in both innate and adaptive immune responses, including evidence for impaired T regulatory function.
Aditi, Sinha, Arvind, Bagga
openaire   +2 more sources

Nephrotic Syndrome

Pediatrics in Review, 2015
On the basis of observational studies, the most common cause of nephrotic syndrome in school-aged children is minimal change disease. On the basis of research evidence and consensus, corticosteroids are considered first-line therapy for treatment of nephrotic syndrome.
Tecile Prince, Andolino   +1 more
openaire   +2 more sources

Nephrotic Syndrome

Pediatric Clinics of North America, 2019
Nephrotic syndrome is characterized by edema, proteinuria, hypoalbuminemia, and hyperlipidemia. Minimal change disease, the most common cause in childhood, generally responds to corticosteroids, although most patients experience disease relapses. Focal segmental glomerulosclerosis is usually resistant to corticosteroids and carries a significant risk ...
Chia-Shi, Wang, Larry A, Greenbaum
openaire   +2 more sources

Nephrotic Syndrome

Primary Care: Clinics in Office Practice, 1979
The nephrotic syndrome appears to be an expression of a number of complex disease processes. The supportive, as well as the definitive, management of the nephrotic syndrome patient requires considerable knowledge of disease pathophysiology, drugs, and drug interactions. Therapy of any degree cannot be undertaken lightly for fear of further compromising
D, Nagar, R L, Wathen
openaire   +2 more sources

Familial nephrotic syndrome

The American Journal of Medicine, 1974
Abstract Systematic pedigree information was obtained from 70 patients with idiopathic nephrotic syndrome; 16 patients were found to have familial nephrotic syndrome including 1 pair of affected monozygotic twins, 5 affected sibling pairs, 2 affected first cousins from a consanguineous family and 2 patients with sporadic cases from consanguineous ...
P I, Bader   +3 more
openaire   +2 more sources

Congenital nephrotic syndrome

Pediatric Nephrology, 2001
A female infant born at 34 weeks' gestation after several days of ruptured membranes had a Potter-like face and compression-induced limb posture consistent with oligohydramnios. Oedema developed on day 2; initial investigations showed massive proteinuria, hypoalbuminaemia, hyponatraemia, acidosis and marked renal insufficiency. The infant was intubated
A, Koziell   +4 more
openaire   +4 more sources

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