Results 161 to 170 of about 167,118 (284)
Iatrogenic atrial dysfunction after prophylactic left atrial appendage exclusion: a proposed post-procedural atrial phenotype. [PDF]
Shuhaiber JH.
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Abstract figure legend Sex‐specific models of the human atrial myocyte in normal sinus rhythm (nSR) and chronic atrial fibrillation (cAF) revealed increased alternans susceptibility in cAF males and DADs in females, driven primarily by ICaL and ryanodine receptor remodelling.
Nathaniel T. Herrera +6 more
wiley +1 more source
The Not-So-Fast Tachycardia: A Paradoxical Rhythm Leading to Cardiomyopathy. [PDF]
Alnazeer M +4 more
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Computational model of haemodynamics during atrial fibrillation
Abstract figure legend We developed a simplified computational model that replicates patient‐specific haemodynamics during both normal sinus rhythm and atrial fibrillation (AF). Our computational model comprises: (1) an electrical subsystem that generates unco‐ordinated atrial and irregular ventricular activation times characteristic of AF and (2) a ...
Felix Plappert +7 more
wiley +1 more source
Recurrent Syncope and Ventricular Standstill Due to Phase 4 His-Purkinje Block. [PDF]
Ahmad F, Lo CCW, Youssef G, Ilsar R.
europepmc +1 more source
Caenorhabditis elegans as an in vivo model system for human inherited primary arrhythmia syndromes
Abstract figure legend Most genes involved in inherited primary arrhythmia syndromes (IPAS) are conserved in Caenorhabditis elegans, where genetic manipulation enables functional characterization of variants, identification of regulatory proteins, and in vivo drug testing.
Antoine Delinière +6 more
wiley +1 more source
A Case of Wide Complex Tachycardia With Right Bundle Branch Block-Electrocardiographic Features to Distinguish Supraventricular Tachycardia With Aberrancy Versus Ventricular Tachycardia. [PDF]
Moe MEE, Loo G, Chong D, Ching KC.
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Abstract figure legend Schematic overview of the temporal evolution of the post‐myocardial infarction substrate leading to ventricular tachycardia. Acute ischaemic injury initiates metabolic and electrophysiological disruption, followed by progressive inflammatory, fibrotic and structural remodelling within the infarct border zone.
Mohammed Obeidat +6 more
wiley +1 more source

