Results 111 to 120 of about 12,044 (250)

Headache Associated with Myasthenia Gravis: The Impact of Mild Ocular Symptoms [PDF]

open access: gold, 2011
Yoshinori Nishimoto   +6 more
openalex   +1 more source

Risk of New‐Onset Myasthenia Gravis Following COVID‐19 Infection and Vaccination: A Population‐Based Case–Control Study

open access: yesEuropean Journal of Neurology, Volume 33, Issue 3, March 2026.
This population‐based case–control study using the Clalit Health Services database (Israel) evaluated adults ≥ 18 years (2020–2022) to assess the risk of new‐onset myasthenia gravis (MG) following confirmed SARS‐CoV‐2 infection or Pfizer‐BioNTech (BNT162b2) vaccination.
Tasneem Kab   +6 more
wiley   +1 more source

Equine botulism

open access: yesEquine Veterinary Journal, Volume 58, Issue 2, Page 333-347, March 2026.
Abstract Botulism is a severe and often fatal disease in equine patients worldwide. Clostridium botulinum is a ubiquitous soil organism which produces a potent neurotoxin resulting in neuromuscular blockade and flaccid paralysis in affected animals. Definitive diagnosis is often impractical or impossible, leading to diagnosis and treatment based on ...
Kali Slavik   +2 more
wiley   +1 more source

Myasthenia Gravis Patients with Anti-MuSK Antibodies [PDF]

open access: yes, 2009
In myasthenia gravis (MG) patients without detectable anti-acetylcholine receptor (anti-AChR) antibody, referred to as seronegative myasthenia gravis patients, there is a variable proportion of patients with antibodies against the muscle-specific kinase (
Davorka Vranješ   +5 more
core   +1 more source

The fc fragment of IgMs binds C1q to activate the first step of the classical complement pathway, while inhibiting complement‐dependent cytotoxicity

open access: yesThe FEBS Journal, Volume 293, Issue 5, Page 1358-1375, March 2026.
Multimeric IgM‐fragment crystallizable region (Fc) fragments retain the ability to bind C1q and initiate the classical complement pathway, leading to C4 activation and deposition in vitro. However, the Fc cores can also inhibit complement‐dependent cytotoxicity by competing with surface‐bound antibodies for C1q engagement.
Andrea J. Pinto   +9 more
wiley   +1 more source

Frontalis sling procedure for ocular myasthenia gravis

open access: yesClinical Ophthalmology, 2012
Shinichi Asamura1, Hirohiko Kakizaki2, Mitsuhiro Enjyo1, Takahiro Hashimoto1, Noritaka Isogai11Department of Plastic and Reconstructive Surgery, Kinki University School of Medicine, Osakasayama, Osaka, Japan; 2Department of Ophthalmology, Aichi Medical ...
Isogai N   +4 more
doaj  

Cranial Nerve Involvement With Diplopia as Presenting Feature of CMT1H Caused by Recurring FBLN5 Variant

open access: yesJournal of the Peripheral Nervous System, Volume 31, Issue 1, March 2026.
ABSTRACT Background and Aims CMT1H is a rare, autosomal dominant, demyelinating subtype of CMT caused by variants in FBLN5. Symptomatic cranial nerve involvement has never been reported in patients with CMT1H. Case Report We report a 45‐year‐old woman with a history of long‐standing diplopia.
Georgios Koutsis   +11 more
wiley   +1 more source

Ocular Myasthenia Gravis and Thymus Gland Hyperplasia–A Case Report

open access: yesTNOA Journal of Ophthalmic Science and Research
Myasthenia gravis (MG) is caused by antibodies directed at acetylcholine receptors or functionally related proteins in the postsynaptic membrane at the neuromuscular junction.
Sujit Das
doaj   +1 more source

بررسی وضعيت سرولوژيک و الکتروفيزيولوژيک بيماران مبتلا به مياستنی گراويس [PDF]

open access: yes, 2013
زمينه و هدف: مياستنی گراويس نوعی اختلال خود ايمنی است که به طور اساسي در اثر آنتی بادی‌های ضد رسپتور استيل کولين در محل اتصال عصب به عضله ايجاد شده و کاهش رسپتورهای مذکور سبب اختلال در انتقال نوروترانسميتر و خستگی و ضعف عضلانی می‌شود.
اميرزرگر, علی اکبر   +6 more
core  

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